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Tobacco Smoking and Atherosclerosis

Pathogenesis and Cellular Mechanisms

  • Book
  • © 1990

Overview

Editors:
  1. John N. Diana

    1. Tobacco and Health Research Institute, University of Kentucky, Lexington, USA

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Part of the book series: Advances in Experimental Medicine and Biology (AEMB, volume 273)

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Table of contents (34 chapters)

  1. Effect of Cigarette Smoking on Platelets, Vascular Tissue and Eicosanoids

    1. Acute Platelet Activation Induced by Smoking Cigarettes: In Vivo and Ex Vivo Studies in Humans

      • Kai G. Schmidt, Jens W. Rasmussen, Vagn Bonnevie-Nielsen
      Pages 199-209

    2. Alterations of Arachidonate Metabolism in Cardiovascular System by Cigarette Smoking

      • Hsin-Hsiung Tai, Wen-Chang Chang, Ying Liu, Shoshi Fukuda
      Pages 211-224

    3. Effect of Nicotine and Carbon Monoxide on Prostacyclin Production by the Rabbit Heart

      • David James Effeney
      Pages 225-235

  2. Effect of Cigarette Smoking on Diet, Cholesterol/Lipoproteins

    1. Relations Between Smoking, Food Intake and Plasma Lipoproteins

      • Anders G. Olsson, Jörgen Mölgaard
      Pages 237-243

    2. Lipoproteins and Diet in the Pathogenesis of Atherosclerosis

      • James W. Anderson, Tammy L. Floore
      Pages 245-258

    3. Apolipoprotein Profiles in Randomly Selected Smokers and Matched Controls

      • Jörgen Mölgaard, Anders G. Olsson
      Pages 259-266

    4. Normalization of High Density Lipoprotein Cholesterol Following Cessation from Cigarette Smoking

      • Robert J. Moffatt
      Pages 267-272

  4. Cigarette Smoking and Coronary Artery Atherosclerosis

    1. Effects of Cigarette Smoking on Coronary Vascular Dynamics: Relationship to Coronary Atherosclerosis

      • Lloyd W. Klein, Annabelle S. Volgman
      Pages 301-310

    2. Cigarette Smoking and Coronary Artery Disease

      • Graham A. Colditz
      Pages 311-326

    3. Adrenal Release of Catecholamines in the Coronary and Myocardial Response to Nicotine

      • H. Fred Downey
      Pages 327-337

    4. Effects of Cigarette Smoke and Nicotine on Platelets and Experimental Coronary Artery Thrombosis

      • J. D. Folts, S. A. Gering, S. W. Laibly, B. G. Bertha, F. C. Bonebrake, J. W. Keller
      Pages 339-358

  5. Atherosclerosis and the Central Nervous System

    1. The Central Nervous System and Atherogenesis: Interrelationships

      • William H. Gutstein, Joseph M. Wu
      Pages 359-380

  6. Back Matter

    Pages 381-393
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Keywords

About this book

Atherosclerosis is the principal underlying cause of cardiovascular and cerebrovascular disease in people of the Western world. Cigarette smoking has been implicated in both the initiation and exacerbation of the atherosclerotic process. Data to support this implication derives primarily from epidemiologic studies where the relationship between the incidence of atherosclerosis in people who smoke cigarettes has been shown to have a strong correlation. There are few well established explanations for this phenomenon, and basic molecular, biochemical, and cellular mechanisms associated with smoking and the development of atherosclerosis remain both undefined and virtually unexplored. Even the epidemiologic correlation between cigarette smoking and the development of atherosclerosis needs further critical studies. It is known that individuals who do not smoke cigarettes develop athero­ sclerosis and it is also known that in people who smoke but have normal or low blood cholesterol/lipoprotein levels, the incidence of development of atherosclerosis is no different from that which is found in a non­ smoking population. Answers which explain such observations must address fundamental biological mechanisms. Toward this end, the purpose of this volume is to assemble, in a single publication, information which will address the questions; what basic cellular and/or molecular mechanisms are associated with the development of atherosclerosis and how does cigarette smoking influence such mechanisms to initiate or exacerbate the atherosclerotic process? Clearly, the development of atherosclerosis is a complex, multifactorial biological event.

Editors and Affiliations

  • Tobacco and Health Research Institute, University of Kentucky, Lexington, USA

    John N. Diana

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