Skip to main content

Silicon in Uraemic Rats

  • Chapter
Nephrotoxicity
  • 12 Accesses

Abstract

Silicon is one of essential trace elements for normal growth, bone formation and development in rats (1) and also plays an important role in other vital processes in animals (2). Silicon is found in brain, liver, lung, kidney (especially in mitochondria) , and in bone, blood and urine.

This is a preview of subscription content, log in via an institution to check access.

Access this chapter

Chapter
USD 29.95
Price excludes VAT (USA)
  • Available as PDF
  • Read on any device
  • Instant download
  • Own it forever
eBook
USD 39.99
Price excludes VAT (USA)
  • Available as EPUB and PDF
  • Read on any device
  • Instant download
  • Own it forever
Softcover Book
USD 54.99
Price excludes VAT (USA)
  • Compact, lightweight edition
  • Dispatched in 3 to 5 business days
  • Free shipping worldwide - see info

Tax calculation will be finalised at checkout

Purchases are for personal use only

Institutional subscriptions

References

  1. Schwarz K, Milne DB. Growth-promoting effects of silicon in rats. Nature, 239: 339, 1972.

    Article  Google Scholar 

  2. Carlisle DM. Silicon: An essential element for the chick. Science, 178: 619, 1972.

    Article  PubMed  CAS  Google Scholar 

  3. Leong ASY. Pathologic findings in silicone spallation: Autopsy and biopsy studies. Annals Academy Med, 12: 304–310, 1983.

    CAS  Google Scholar 

  4. Laohapand T, Osman EM, Morley AR, Word MK, Kerr DNS. Accumulation of silicon elaster in regular dialysis. Proc. EDTA. 19:143, 1982.

    Google Scholar 

  5. Hauglustaine D, Van Damme B, Daeness P, Michielsen P. Silicon nephropathy: a possible occupational hazard. Nephron. 26: 219, 1980.

    Article  PubMed  CAS  Google Scholar 

  6. Hershey CO, Ricantai ES, Hershey LA, Varnes AW, Lavin PJM, Strain WH. Silicon as a potential uremic neurotoxin: trace element analysis in patients with renal failure. Neurology, 33: 786, 1986.

    Article  Google Scholar 

  7. Hosokawa S, Morinaga M, Nishitani H, Maeda T, Yoshida O. Silicon in chronic hemodialysis patients. ASAIO transaction, 33: 1987 (in press).

    Google Scholar 

  8. Berlyne GM, Adler AJ, Ferran N, Bennett S, Holt J. Silicon metabolism. Nephron. 44:36, 1986.

    Article  PubMed  Google Scholar 

  9. Mauras Y, Riberi P, Cartier F, Allain P. Increase in blood silicon concentration in patients with renal failure. Biomedicine, 33:228, 1980.

    PubMed  CAS  Google Scholar 

  10. Berlyne GM, Dudek E, Adler AJ, Rubin JE, Seidman M. Silicon metabolism: the basic facts in renal failure. Kidney Int. 28 suppl 17: 175, 1985.

    Google Scholar 

  11. Goligorsdy MS, Chaimovilz Cidio, Nir Y, Rapopert J, Kol R, Yehuda J. X-ray microanalysis of uremic nephrocalcinosis: Cellular distribution of calcium, aluminium and silicon in uremic nephrocalcinosis. Mineral Electrolyte Metab. 11:301, 1985.

    Google Scholar 

Download references

Author information

Authors and Affiliations

Authors

Editor information

Editors and Affiliations

Rights and permissions

Reprints and permissions

Copyright information

© 1989 Springer Science+Business Media New York

About this chapter

Cite this chapter

Hosokawa, S., Morinaga, M., Nishitani, H., Yoshida, O. (1989). Silicon in Uraemic Rats. In: Bach, P.H., Lock, E.A. (eds) Nephrotoxicity. Springer, Boston, MA. https://doi.org/10.1007/978-1-4757-2040-2_96

Download citation

  • DOI: https://doi.org/10.1007/978-1-4757-2040-2_96

  • Publisher Name: Springer, Boston, MA

  • Print ISBN: 978-1-4757-2042-6

  • Online ISBN: 978-1-4757-2040-2

  • eBook Packages: Springer Book Archive

Publish with us

Policies and ethics