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Klinische Neuropathologie des Parkinson-Syndroms — Proteintyp und Dopamintyp der Demelaninisierung

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Parkinson plus

Zusammenfassung

Die neuronale Zytopathologie des Parkinsonismus interessiert ätiopathogenetisch besonders im Hinblick auf morphologisch-biochemische Strukturbeziehungen. Demgegenüber ist ihre zentralnervöse Topologie für die klinisch-neurologische Symptombildung von Bedeutung.

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Authors
  1. H. Jacob
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  2. T. Kawagoe
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Editors and Affiliations

  1. Zentrum der Neurologie und Neurochirurgie, Klinikum der Universität Frankfurt, Schleusenweg 2-16, 6000, Frankfurt/M. 71, Deutschland

    Peter-A. Fischer (Leiter der Abteilung für Neurologie) (Leiter der Abteilung für Neurologie)

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© 1984 Springer-Verlag Berlin-Heidelberg

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Jacob, H., Kawagoe, T. (1984). Klinische Neuropathologie des Parkinson-Syndroms — Proteintyp und Dopamintyp der Demelaninisierung. In: Fischer, PA. (eds) Parkinson plus. Springer, Berlin, Heidelberg. https://doi.org/10.1007/978-3-642-69839-2_3

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  • DOI: https://doi.org/10.1007/978-3-642-69839-2_3

  • Publisher Name: Springer, Berlin, Heidelberg

  • Print ISBN: 978-3-540-13566-1

  • Online ISBN: 978-3-642-69839-2

  • eBook Packages: Springer Book Archive

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