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Symptoms and Signs of Hemorrhagic Stroke

  • Seung-Hoon LeeEmail author
Chapter
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Part of the Stroke Revisited book series (STROREV)

Abstract

In patients with hemorrhagic stroke, immediate diagnosis is performed based on symptoms, signs, and brain imaging. Clinical symptoms are likely to worsen during the first 24–72 h after onset, and emergency care should be initiated immediately after establishing the diagnosis. Intracerebral hemorrhage (ICH) can present with general symptoms such as headache, vomiting, and altered consciousness and with focal neurologic deficits according to the ICH location – lobar, basal ganglia, thalamus, cerebellum, and brain stem. Headaches are also the most common symptom in subarachnoid hemorrhage but present with a higher intensity than do headaches caused by intracerebral hemorrhage. In addition, subarachnoid hemorrhage causes altered consciousness, and seizures occur at a higher frequency. In this chapter, the clinical symptoms and signs of hemorrhagic stroke will be discussed in detail.

Keywords

Altered Consciousness Intracerebral Haemorrhage (ICH) Basal Ganglia Hemorrhage Caudate Hemorrhage Lobar Hemorrhage 
These keywords were added by machine and not by the authors. This process is experimental and the keywords may be updated as the learning algorithm improves.

References

  1. 1.
    Kase CS, Williams JP, Wyatt DA, et al. Lobar intracerebral hematomas: clinical and CT analysis of 22 cases. Neurology. 1982;32:1146–50.CrossRefPubMedGoogle Scholar
  2. 2.
    Kawahara N, Sato K, Muraki M, et al. CT classification of small thalamic hemorrhages and their clinical implications. Neurology. 1986;36:165–72.CrossRefPubMedGoogle Scholar
  3. 3.
    Barraquer-Bordas L, Illa I, Escartin A, Ruscalleda J, et al. Thalamic hemorrhage. A study of 23 patients with diagnosis by computed tomography. Stroke. 1981;12:524–7.CrossRefPubMedGoogle Scholar
  4. 4.
    Berlit P, Tornow K. Outcome of intracerebral hemorrhage: clinical and CT findings in 326 patients. Eur J Neurol. 1994;1:29–34.CrossRefPubMedGoogle Scholar
  5. 5.
    Miranpuri AS, Aktüre E, Baggott CD, et al. Demographic, circadian, and climatic factors in non-aneurysmal versus aneurysmal subarachnoid hemorrhage. Clin Neurol Neurosurg. 2013;115:298–303.CrossRefPubMedGoogle Scholar
  6. 6.
    Linn FH, Wijdicks EF, van der Graaf Y, et al. Prospective study of sentinel headache in aneurysmal subarachnoid haemorrhage. Lancet. 1994;344:590–3.CrossRefPubMedGoogle Scholar
  7. 7.
    Hijdra A, van Gijn J. Early death from rupture of an intracranial aneurysm. J Neurosurg. 1982;57:765–8.CrossRefPubMedGoogle Scholar
  8. 8.
    Vermeulen M, van Gijn J, Hijdra A, et al. Causes of acute deterioration in patients with a ruptured intracranial aneurysm. A prospective study with serial CT scanning. J Neurosurg. 1984;60:935–9.CrossRefPubMedGoogle Scholar
  9. 9.
    Pinto AN, Canhao P, Ferro JM. Seizures at the onset of subarachnoid haemorrhage. J Neurol. 1996;243:161–4.CrossRefPubMedGoogle Scholar
  10. 10.
    Liu Q, Ding YH, Zhang JH, et al. ECG change of acute subarachnoid hemorrhagic patients. Acta Neurochir Suppl. 2011;111:357–9.CrossRefPubMedGoogle Scholar
  11. 11.
    Laun A, Tonn JC. Cranial nerve lesions following subarachnoid hemorrhage and aneurysm of the circle of Willis. Neurosurg Rev. 1988;11:137–41.CrossRefPubMedGoogle Scholar

Copyright information

© Springer Science+Business Media Singapore 2018

Authors and Affiliations

  1. 1.Department of NeurologySeoul National University HospitalSeoulSouth Korea
  2. 2.Korean Cerebrovascular Research InstituteSeoulSouth Korea

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