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Ventricular Arrhythmias in the First 15 Minutes of Acute Regional Myocardial Ischemia in the Isolated Pig Heart: Possible Role of Injury Currents

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Book cover Sudden Death

Part of the book series: Developments in Cardiovascular Medicine ((DICM,volume 4))

Abstract

There is a great deal of experimental evidence suggesting that ventricular arrhythmias in myocardial infarction are due to reentry (1–5). Single premature beats, multiple premature beats, ventricular tachycardia and ventricular fibrillation could in principle all be caused by reentrant mechanisms. However, the precise pathways of excitation during such arrhythmias have not been completely delineated. Furthermore, programmed stimulation performed in patients with ventricular tachycardia during the first 24 hours of myocardial infarction revealed that the tachycardia could not be initiated by one, two or even three electrically induced premature beats, and it was suggested that at least some of the early arrhythmias might not be due to reentry but to some sort of automaticity (6).

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© 1980 Martinus Nijhoff Publishers bv, The Hague

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Janse, M.J., Morsink, H., van Capelle, F.J.L., Kléber, A.G., Wilms-Schopman, F., Durrer, D. (1980). Ventricular Arrhythmias in the First 15 Minutes of Acute Regional Myocardial Ischemia in the Isolated Pig Heart: Possible Role of Injury Currents. In: Kulbertus, H.E., Wellens, H.J.J. (eds) Sudden Death. Developments in Cardiovascular Medicine, vol 4. Springer, Dordrecht. https://doi.org/10.1007/978-94-009-8834-7_6

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  • DOI: https://doi.org/10.1007/978-94-009-8834-7_6

  • Publisher Name: Springer, Dordrecht

  • Print ISBN: 978-94-009-8836-1

  • Online ISBN: 978-94-009-8834-7

  • eBook Packages: Springer Book Archive

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