Professor Forfar’s wide ranging review of Rhesus disease left out one aspect — something that has haunted me personally for some fifteen years. One of my own first patients with Rhesus antibodies and with complications, including anti-C, had a very high bilirubin. I had expected that an exchange transfusion would be carried out. But, the hospital — a university children’s hospital — advised that it was their practice to look only at the bilirubin. In this instance it was not indirect and not dangerous and they would not do an exchange transfusion for this child.
KeywordsBrain Damage Exchange Transfusion Maternity Hospital Haemolytic Uraemic Syndrome Calcium Gluconate
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