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Enough has been Written About the Treatment of ALI, but has Enough been Said About how to Prevent It?

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Practical Issues in Anesthesia and Intensive Care 2013

Abstract

Acute Lung Injury (ALI) is a syndrome characterized by the presence of non-cardiogenic pulmonary edema and hypoxemia as a result of various types of disease or condition.

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References

  1. Bernard GR, Artigos A, Brigham Kl et al (1994) The American European consensus conference on ARDS: definitions, mechanism, relevant outcomes and clinical trial coordination. Am J Resp Care Med 149:818–824

    Google Scholar 

  2. Ferguson ND, Frutos F, Esteban A et al (2007) Clinical risk conditions for ALI in the intensive care unit and Hospital ward: a prospective observational study. Crit Care 11:R96

    Google Scholar 

  3. Galvin I, Ferguson ND (2011) Acute Lung Injury in the ICU: focus on prevention annual update in intensive care and emergency medicine. In: Vincent JL (ed) pp 117–128

    Google Scholar 

  4. Wrigge H, Uhlig U, Zinserling J et al (2004) The effects of different ventilator settings on pulmonary and systemic inflammation responses during major surgery. Anesth Analg 98:775–781

    Article  PubMed  Google Scholar 

  5. Michelet P, D’Joumo XP, Roch A et al (2006) Protective ventilation influence systemic inflammation after esophagectomy. Anesthesiology 105:911–919

    Google Scholar 

  6. Hedenstierna G, Edmark L (2005) The effects of anesthesia and muscle paralysis on the respiratory system. Intens Care Med 31:1327–1335

    Article  Google Scholar 

  7. Marshall BE (1998) Effects of anesthetics on pulmonary gas exchange. In: Stanley TH, Sperry RJ (ed) Anesthesia and the Lung. Kluwer, London, pp 117–125

    Google Scholar 

  8. Lindberg P, Gunnarsson L, Tokics L et al (1992) Atelectasis, gas exchange and lung function in the postoperative period. Acta Anaesth Scand 36:546–553

    Article  PubMed  CAS  Google Scholar 

  9. Brismar B, Hedenstierna G, Lundquist H et al (1985) Pulmonary densities during anesthesia with muscular relaxation: a proposal of atelectasis. Anesthesiology 62:422–428

    Article  PubMed  CAS  Google Scholar 

  10. Rothen HU, Sporre B, Engberg G et al (1993) Reexpansion of atelectasis during general anesthesia: a computed tomography study. Br J Anesth 71:788–795

    Article  CAS  Google Scholar 

  11. Rothen HU, Sporre B, Engberg G et al (1995) Influence of gas composition on recurrence of atelectasis after a reexpansion maneuver during general anesthesia. Anesthesilogy 82:832–842

    Article  CAS  Google Scholar 

  12. Schultz MJ, Hitsma JJ, Slutsky AS et al (2007) What TV should be used in patients without ALI? Anesthesiology 106:1226–1231

    Google Scholar 

  13. Iscimen R, Cartu R, Ylmer M et al (2008) Risk factors for the development of ALI in patients with septic shock: an observational cohort study. Crit Care Med 36:1518–1522

    Article  PubMed  Google Scholar 

  14. The National Heart (2006) Lung and blood institute ARDS clinical trials network comparison of two fluid-management strategies in ALI. N Engl J Med 354:2564–2576

    Google Scholar 

  15. Jia X, Malhorta A, Saeed M et al (2008) Risk factors for ARDS in patients without ARDS. Chest 133:853–861

    Article  PubMed  Google Scholar 

  16. Huges CG, Weavind L, Banerjee A et al (2010) Intraoperative risk factors for ARDS in critically ill patients. Anesth Analg 111:464–467

    Article  Google Scholar 

  17. Popovsky MA, Abel MP, Moore SB (1983) Transfusion related acute lung injury associated with passive transfer of antileukocyte antibodies. Am Rev Resp Dis 118:185–189

    Google Scholar 

  18. Kahn H, Belshar J, Yilmer M et al (2007) FFP and platelet transfusion are associated with development of ALI in critically ill medical patients. Chest 131:1308–1314

    Article  Google Scholar 

  19. Hume HA (2009) TRALI: moving toward prevention. Transfusion 49:402–405

    Google Scholar 

  20. Murphy DJ, Howard D, Muriithi A et al (2009) Red blood cell transfusion practices in ALI: what do patient factors contribute? Crit Care Med 36:1935–1940

    Article  Google Scholar 

  21. Ylmar M, Keegan MT, Iscimen R et al (2007) Protocol-guided limitation of large TV ventilation and inappropriate transfusion. Crit Care 35:1660–1666

    Article  Google Scholar 

  22. Moss M, Guidot DM, Steinberg KP et al (2000) Diabetic patients have a decreased incidence of ARDS. Crit Care Med 28:2187–2192

    Article  PubMed  CAS  Google Scholar 

  23. Iscimen R, Cartin-Ceba R, Ylmar M et al (2008) Risk factors for the development of ALI in patients with septic shock: an observational cohort study. Crit Care Med 36:1518–1522

    Article  PubMed  Google Scholar 

  24. HagiwareS, Iwasoka H, Hazegawa a et al (2008) Effects of hyperglycemia and insulin therapy on high mobility group box 1 in endotoxin-induced ALI in a rat model. Crit Care Med 36:2407–2413

    Google Scholar 

  25. Ramasamy R, Yan SF, Herold K et al (2008) Receptor for advanced glycation end products: fundamental roles in the inflammatory response: winding the way to the pathogenesis of endothelial disfunction and atherosclerosis. Ann NY Scand Sci 1126:7–18

    Google Scholar 

  26. Calfec CS, Ware LB, Eisner MD et al (2008) Plasma receptor for AGE products and clinical outcome in ALI. Thorax 63:1083–1089

    Article  Google Scholar 

  27. Schnapp LM, Donhoe S, Chen J et al (2006) Missing ARDS proteome: identification of the insulin-like growth factor (IGF)/IGF binding protein-3 pathway in ALI. Am J Pathol 169:86–95

    Article  PubMed  CAS  Google Scholar 

  28. Pirat A, Zeyneloghn P, Aldemir D et al (2006) Pretreatment with simvastin reduces lung injury related to intestinal ischemia reperfusion in rats. Anesth Analg 102:225–232

    Article  PubMed  CAS  Google Scholar 

  29. Dietch EA (2001) Role of gut lymphatic in MOF. Curr Opin Crit Care 7:92–98

    Google Scholar 

  30. Galban C, Montejo JC, Mesejo A et al (2000) An immune-enhancing enteral diet reduces mortality rate and episodes of bacteremia in septic intensive care unit patients. Crit Care Med 28:643–648

    Google Scholar 

  31. Wrigge H, Zinserling J, Stüber F et al (2000) Effects of mechanical ventilation on release of cytokines into systemic circulation in patients with normal pulmonary function. Anesthesiology 93:1413–1417

    Article  PubMed  CAS  Google Scholar 

  32. Honiden S, Gong M (2009) Diabetes, insulin, and development of ALI. Crit Care Med 37:2455–2464

    Article  PubMed  CAS  Google Scholar 

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Correspondence to Biagio Allaria .

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Allaria, B. (2014). Enough has been Written About the Treatment of ALI, but has Enough been Said About how to Prevent It?. In: Allaria, B. (eds) Practical Issues in Anesthesia and Intensive Care 2013. Springer, Milano. https://doi.org/10.1007/978-88-470-5529-2_2

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  • DOI: https://doi.org/10.1007/978-88-470-5529-2_2

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  • Publisher Name: Springer, Milano

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  • Online ISBN: 978-88-470-5529-2

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