Abstract
Acute Lung Injury (ALI) is a syndrome characterized by the presence of non-cardiogenic pulmonary edema and hypoxemia as a result of various types of disease or condition.
Access this chapter
Tax calculation will be finalised at checkout
Purchases are for personal use only
References
Bernard GR, Artigos A, Brigham Kl et al (1994) The American European consensus conference on ARDS: definitions, mechanism, relevant outcomes and clinical trial coordination. Am J Resp Care Med 149:818–824
Ferguson ND, Frutos F, Esteban A et al (2007) Clinical risk conditions for ALI in the intensive care unit and Hospital ward: a prospective observational study. Crit Care 11:R96
Galvin I, Ferguson ND (2011) Acute Lung Injury in the ICU: focus on prevention annual update in intensive care and emergency medicine. In: Vincent JL (ed) pp 117–128
Wrigge H, Uhlig U, Zinserling J et al (2004) The effects of different ventilator settings on pulmonary and systemic inflammation responses during major surgery. Anesth Analg 98:775–781
Michelet P, D’Joumo XP, Roch A et al (2006) Protective ventilation influence systemic inflammation after esophagectomy. Anesthesiology 105:911–919
Hedenstierna G, Edmark L (2005) The effects of anesthesia and muscle paralysis on the respiratory system. Intens Care Med 31:1327–1335
Marshall BE (1998) Effects of anesthetics on pulmonary gas exchange. In: Stanley TH, Sperry RJ (ed) Anesthesia and the Lung. Kluwer, London, pp 117–125
Lindberg P, Gunnarsson L, Tokics L et al (1992) Atelectasis, gas exchange and lung function in the postoperative period. Acta Anaesth Scand 36:546–553
Brismar B, Hedenstierna G, Lundquist H et al (1985) Pulmonary densities during anesthesia with muscular relaxation: a proposal of atelectasis. Anesthesiology 62:422–428
Rothen HU, Sporre B, Engberg G et al (1993) Reexpansion of atelectasis during general anesthesia: a computed tomography study. Br J Anesth 71:788–795
Rothen HU, Sporre B, Engberg G et al (1995) Influence of gas composition on recurrence of atelectasis after a reexpansion maneuver during general anesthesia. Anesthesilogy 82:832–842
Schultz MJ, Hitsma JJ, Slutsky AS et al (2007) What TV should be used in patients without ALI? Anesthesiology 106:1226–1231
Iscimen R, Cartu R, Ylmer M et al (2008) Risk factors for the development of ALI in patients with septic shock: an observational cohort study. Crit Care Med 36:1518–1522
The National Heart (2006) Lung and blood institute ARDS clinical trials network comparison of two fluid-management strategies in ALI. N Engl J Med 354:2564–2576
Jia X, Malhorta A, Saeed M et al (2008) Risk factors for ARDS in patients without ARDS. Chest 133:853–861
Huges CG, Weavind L, Banerjee A et al (2010) Intraoperative risk factors for ARDS in critically ill patients. Anesth Analg 111:464–467
Popovsky MA, Abel MP, Moore SB (1983) Transfusion related acute lung injury associated with passive transfer of antileukocyte antibodies. Am Rev Resp Dis 118:185–189
Kahn H, Belshar J, Yilmer M et al (2007) FFP and platelet transfusion are associated with development of ALI in critically ill medical patients. Chest 131:1308–1314
Hume HA (2009) TRALI: moving toward prevention. Transfusion 49:402–405
Murphy DJ, Howard D, Muriithi A et al (2009) Red blood cell transfusion practices in ALI: what do patient factors contribute? Crit Care Med 36:1935–1940
Ylmar M, Keegan MT, Iscimen R et al (2007) Protocol-guided limitation of large TV ventilation and inappropriate transfusion. Crit Care 35:1660–1666
Moss M, Guidot DM, Steinberg KP et al (2000) Diabetic patients have a decreased incidence of ARDS. Crit Care Med 28:2187–2192
Iscimen R, Cartin-Ceba R, Ylmar M et al (2008) Risk factors for the development of ALI in patients with septic shock: an observational cohort study. Crit Care Med 36:1518–1522
HagiwareS, Iwasoka H, Hazegawa a et al (2008) Effects of hyperglycemia and insulin therapy on high mobility group box 1 in endotoxin-induced ALI in a rat model. Crit Care Med 36:2407–2413
Ramasamy R, Yan SF, Herold K et al (2008) Receptor for advanced glycation end products: fundamental roles in the inflammatory response: winding the way to the pathogenesis of endothelial disfunction and atherosclerosis. Ann NY Scand Sci 1126:7–18
Calfec CS, Ware LB, Eisner MD et al (2008) Plasma receptor for AGE products and clinical outcome in ALI. Thorax 63:1083–1089
Schnapp LM, Donhoe S, Chen J et al (2006) Missing ARDS proteome: identification of the insulin-like growth factor (IGF)/IGF binding protein-3 pathway in ALI. Am J Pathol 169:86–95
Pirat A, Zeyneloghn P, Aldemir D et al (2006) Pretreatment with simvastin reduces lung injury related to intestinal ischemia reperfusion in rats. Anesth Analg 102:225–232
Dietch EA (2001) Role of gut lymphatic in MOF. Curr Opin Crit Care 7:92–98
Galban C, Montejo JC, Mesejo A et al (2000) An immune-enhancing enteral diet reduces mortality rate and episodes of bacteremia in septic intensive care unit patients. Crit Care Med 28:643–648
Wrigge H, Zinserling J, Stüber F et al (2000) Effects of mechanical ventilation on release of cytokines into systemic circulation in patients with normal pulmonary function. Anesthesiology 93:1413–1417
Honiden S, Gong M (2009) Diabetes, insulin, and development of ALI. Crit Care Med 37:2455–2464
Author information
Authors and Affiliations
Corresponding author
Editor information
Editors and Affiliations
Rights and permissions
Copyright information
© 2014 Springer-Verlag Italia
About this chapter
Cite this chapter
Allaria, B. (2014). Enough has been Written About the Treatment of ALI, but has Enough been Said About how to Prevent It?. In: Allaria, B. (eds) Practical Issues in Anesthesia and Intensive Care 2013. Springer, Milano. https://doi.org/10.1007/978-88-470-5529-2_2
Download citation
DOI: https://doi.org/10.1007/978-88-470-5529-2_2
Published:
Publisher Name: Springer, Milano
Print ISBN: 978-88-470-5528-5
Online ISBN: 978-88-470-5529-2
eBook Packages: MedicineMedicine (R0)