Abstract
The tricuspid valve becomes frequently affected in the course of rheumatic heart disease but has often been overlooked. Morphological distortion and dysfunction of the mitral and aortic valves dominate the clinical picture in the rheumatic disease; consequently, most of the attention has been focused on the left-sided valves. Sixty years after open-heart surgery introduction, the timing of surgery and the optimal operation for tricuspid valve disease continues as a matter for discussion in most of international meetings.
Rheumatic tricuspid valve dysfunction is due to stenosis, regurgitation, or mixed lesion. Autopsy studies indicate that the tricuspid valve shows similar morphological abnormalities than the mitral valve. Rheumatic involvement of the tricuspid valve is a combination of leaflet fibrosis or calcified thickening, commissural fusion, and subvalvular apparatus shortening due to fibrosis, shortening with the result of a significant valve mobility restriction producing regurgitation and/or stenosis.
In general, tricuspid valve repair is a long-term stable operation; however, intrinsic valve or iatrogenic residual dysfunctions requiring reoperation are associated with a high hospital mortality and poor prognosis.
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Acknowledgements
JMI was supported by grant CGL2008-04559/BOS from the “Ministerio de Educación y Ciencia,” Spain.
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Revuelta, J.M., Icardo, J.M. (2014). The Rheumatic Tricuspid Valve. In: Giamberti, A., Chessa, M. (eds) The Tricuspid Valve in Congenital Heart Disease. Springer, Milano. https://doi.org/10.1007/978-88-470-5400-4_13
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DOI: https://doi.org/10.1007/978-88-470-5400-4_13
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