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Pathophysiology and Monitoring of Cerebral Edema

  • F. Della Corte
  • O. Piazza
  • A. Clemente
  • M. A. Pennisi

Abstract

After primary neurologic damage due to a traumatic, ischemic, hypoxic or metabolic event, a secondary lesion may arise, worsening it and becoming a vicious circle. Locally, the initial energetic breakdown or the tissue mechanical disruption set off a number of biochemical chain reactions leading to a derangement of molecular structures and liberating toxic mediators. Globally, these reactions are responsible for brain edema and disorders of microcirculation (1).

Keywords

Cerebral Blood Flow Brain Edema Cerebral Edema Cerebral Perfusion Pressure Blood Flow Velocity 
These keywords were added by machine and not by the authors. This process is experimental and the keywords may be updated as the learning algorithm improves.

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Copyright information

© Springer-Verlag Italia, Milano 1996

Authors and Affiliations

  • F. Della Corte
  • O. Piazza
  • A. Clemente
  • M. A. Pennisi

There are no affiliations available

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