Abstract
Muscle involvement in severe disease is known since the dawn of medicine [1], however only in recent years has research focused on this topic. Basic and clinical scientists have made available a rich harvest of clinical, physiological, biochemical and pathological data, and with them the Babel tower problem: “Clinicians face the problem of muscle wasting and weakness; biochemists are confronted with muscle glutamine efflux, physiologists with reduced or absent excitability, pathologists with atrophy and necrosis”. Are they simply the same problem seen through different lenses? Do they perfectly overlap, so that clinically observed muscle wasting corresponds to histologically proven myopathy?
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Latronico, N., Candiani, A. (1999). Muscular Wasting as a Consequence of Sepsis. In: Gullo, A. (eds) Anaesthesia, Pain, Intensive Care and Emergency Medicine — A.P.I.C.E.. Springer, Milano. https://doi.org/10.1007/978-88-470-2145-7_51
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DOI: https://doi.org/10.1007/978-88-470-2145-7_51
Publisher Name: Springer, Milano
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