Topographic Peculiarity in Spastic Changes of the Cerebral Artery After Subarachnoid Hemorrhage
Clinical observations have suggested that delayed ischemic neurological deficit caused by cerebral vasospasm is not always correlated with the location of spasm as revealed by angiography. Recent investigations have indicated an important role for arachidonic acid metabolites, especially prostacycline and thromboxane A2, in the genesis of cerebral vasospasm following subarachnoid hemorrhage [7, 10].
KeywordsSubarachnoid Hemorrhage Basilar Artery Temporal Cortex Cerebral Vasospasm Posterior Inferior Cerebellar Artery
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