Abstract
Clinical observations have suggested that delayed ischemic neurological deficit caused by cerebral vasospasm is not always correlated with the location of spasm as revealed by angiography. Recent investigations have indicated an important role for arachidonic acid metabolites, especially prostacycline and thromboxane A2, in the genesis of cerebral vasospasm following subarachnoid hemorrhage [7, 10].
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References
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© 1988 Springer Japan
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Nakamura, S., Tsubokawa, T., Yoshida, K. (1988). Topographic Peculiarity in Spastic Changes of the Cerebral Artery After Subarachnoid Hemorrhage. In: Suzuki, J. (eds) Advances in Surgery for Cerebral Stroke. Springer, Tokyo. https://doi.org/10.1007/978-4-431-68314-8_133
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DOI: https://doi.org/10.1007/978-4-431-68314-8_133
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