Recovery of Cortical Evoked Potential and LCGU Following Transient MCA Occlusion in the Rat
Recent evidence indicates that the development of infarct is a function of residual blood flow and duration of ischemia [3, 4]. However, it is difficult to predict whether ischemic tissue is viable or not in the early postischemic period. The present investigation was designed to study the relationship of somatosensory evoked potential (SEP), local cerebral glucose utilization (LCGU), and histopathological changes in the transiently MCA-occluded rat.
KeywordsMiddle Cerebral Artery Occlusion Sensorimotor Cortex Somatosensory Evoke Poten Ischemic Brain Damage Ischemic Core
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