Abstract
During the last decade several studies of cerebral blood flow (CBF) and metabolism in the acute phase of head injury have been published. It is the aim of this review to describe the dynamic changes in CBF, cerebral metabolic rate of oxygen (CMRO2), cerebral autoregulation (CA), and reactivity to PaCO2 and barbiturate (metabolic reactivity) in the acute phase after severe head injury and to discuss the therapeutical consequences with reference to prolonged artificial hyperventilation, hypothermia, barbiturate sedation, and mannitol therapy.
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© 1990 Springer-Verlag/Wien
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Cold, G.E. (1990). Summary. In: Cerebral Blood Flow in Acute Head Injury. Acta Neurochirurgica, vol 49. Springer, Vienna. https://doi.org/10.1007/978-3-7091-9101-9_8
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DOI: https://doi.org/10.1007/978-3-7091-9101-9_8
Publisher Name: Springer, Vienna
Print ISBN: 978-3-7091-9103-3
Online ISBN: 978-3-7091-9101-9
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