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Is acute ataxic neuropathy a distinct entity? Clinical-electrophysiological and morphological study

  • A. B. Taly
  • A. Prasad
  • A. Vasanth
  • S. K. Shankar
  • D. Nagaraj
Conference paper

Summary

Ten patients of acute severe sensory ataxic neuropathy seen over 6 years are reported. Presence of severe sensory deficits, absence of ophthalmoplegia or significant motor weakness, distinct electrophysiological abnormalities, frequently persisting and disabling ataxia distinguish this rare disorder from the more benign Miller Fisher syndrome and more commonly observed G. B. Syndrome with residual ataxia.

Keywords

Dorsal Root Ganglion Sural Nerve Axonal Damage Miller Fisher Syndrome Abnormal Heart Rate 
These keywords were added by machine and not by the authors. This process is experimental and the keywords may be updated as the learning algorithm improves.

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References

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    Richter RB (1962) The ataxic form of polyradiculoneuritis (Landry Guillain-Barré Syndrome): clinical and pathological observation. J Neuropathol Exp Neurol 21: 171–184PubMedCrossRefGoogle Scholar
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    Sobue G, Sneda Y, Matsuoka Y, Sobue I (1983) Sensory ataxia: a residual disability of Guillain-Barré Syndrome. Arch Neurol 40: 86–89PubMedCrossRefGoogle Scholar
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    Sterman AB, Schaumburg HH, Asbury AK (1980) The acute sensory neuronopathy syndrome: a distinct clinical entity. Ann Neurol 7: 354–358PubMedCrossRefGoogle Scholar

Copyright information

© Springer-Verlag Wien 1995

Authors and Affiliations

  • A. B. Taly
    • 1
  • A. Prasad
    • 1
  • A. Vasanth
    • 1
  • S. K. Shankar
    • 2
  • D. Nagaraj
    • 1
  1. 1.Department of NeurologyNational Institute of Mental Health and Neuro SciencesBangaloreIndia
  2. 2.Department of NeuropathologyNational Institute of Mental Health and Neuro SciencesBangaloreIndia

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