Cerebral vasospasm after SAH
The introduction and worldwide application of the concept of early surgery after the rupture of an intracranial aneurysm as well as an improved intensive care management in the last two decades have contributed to a significant reduction of morbidity and mortality due to vasospasm. Fisher, Roberson and Ojemann (1977) found that 50% of 50 patients, who had survived SAH caused by aneurysm rupture, developed delayed neurological deficits as a consequence of vasospasm. In the Cooperative Study of Kassell et al. (1990a;b), 7.2% mortality and 6.3% permanent impairments were attributed to vasospasm. In the manifold literature on this topic, data on the incidence of vasospasm and the frequency of associated delayed ischemic deficits vary considerably. Various definitions of vasospasm and differences between the methods used to measure vasospasm are also responsible for the discrepancies.
KeywordsBlood Flow Velocity Intracranial Aneurysm Glasgow Outcome Scale Cerebral Vasospasm Aneurysm Rupture
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