Immunological Response in Early Brain Injury After SAH
This study summarized the role of inflammation in the early brain injury after subarachnoid hemorrhage. Elevation of cytokines, activation of MMPs and phosphorylation of MAPK contributes to neuronal apoptosis and brain edema. Anti-inflammation may be potential strategy for the prevention and suppression of early brain injury after subarachnoid hemorrhage.
KeywordsEarly brain injury Immune Inflammation
This study was partially supported by grants (NS053407) from the National Institutes of Health to J.H.Z.
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