Abstract
Arterial obstruction by atherosclerosis may be improved by certain changes within the vessel wall. These include a reduction in the size of the lesion, senile dilatation of the vessel wall and enlargement of anastomotic channels.
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References
Adams, C. W. M., and Bayliss, O. B. (1976a): Detection of macrophages in atherosclerotic lesions with cytochrome oxidase. Brit. J. Exp. Pathol. 57, 30 to 36.
Adams, C. W. M., and Bayliss, O. B. (1976b): Succinic dehydrogenase and cytochrome oxidase in arterial, venous and other smooth muscle. Atherosclerosis 23, 367 to 370.
Adams, C. W. M., Knox, J., and Morgan, R. S. (1975): The resorption rate of atheroma lipids in situ and implanted subcutaneously. Atherosclerosis 22, 79 to 90.
Adams, C. W. M., Morgan, R. S., and Bayliss, O. B. (1973): No regression of atheroma over one year in rabbits previously fed a cholesterol or enriched diet. Atherosclerosis 18, 429 to 444.
Adams, C. W. M., Knox, J., and Morgan, R. S. (1975): Exchange of plasma radioactive cholesterol with atheroma lipids in situ and implanted subcutaneously. Atherosclerosis 22, 229 to 240.
Adams, C. W. M., Bayliss, O. B., and Turner, D. R. (1975): Phagocytes, lipid-removal and regression of atheroma. J. Pathol. 116, 225 to 238.
Armstrong, J. L., and Megan, M. B. (1972): Lipid depletion in atheromatous coronary arteries in rhesus monkeys after regression diets. Circulat. Res. 30, 675 to 680.
Aschoff, L. (1924): Lectures on Pathology, pp. 131 to 153, Hoeber, New York.
Bortz, W. M. (1968): Reversibility of atherosclerosis in cholesterolfed rabbits. Circulat. Res. 22, 135 to 139.
Clarkson, T. B., King, J. S., Jr., Lofland, H. B., Feldner, M. A., and Bullock, B. C. (1973): Patholigic characteristics and composition of diet-aggravated atherosclerotic plaques during “regression”. Exp. molec. Pathol. 19, 267 to 283.
Daoud, A. S., Jarmolych, J., Augustyn, J. M., Fritz, K. E., Singh, J. K., and Lee, K. T. (1976): Regression of advanced atherosclerosis in swine. Arch. Pathol. Lab. Med. 100, 372 to 379.
French, J. E. (1966): Atherosclerosis in relation to the structure and function of the arterial intima, with special reference to the endothelium. Internat. Rev. exp. Pathol. 5, 253 to 353.
French, J. E., Jennings, M. A., and Florey, H. W. (1965): Morphological studies in atherosclerosis in swine. Ann. N.Y. Acad. Sci. 127, 780 to 799.
Horlick, L., and Katz, L. N. (1949): Retrogression of atherosclerotic lesions on cessation of cholesterol feeding in the chick. J. Lab. Clin. Med. 34, 1427 to 1429.
Miller, G. J., and Miller, N. E. (1975): Plasma-high-density-liproprotein concentration and development of ischaemic heart disease. Lancet 1, 16 to 19.
Peterson, J. E., and Hirst, A. E. (1951): Studies on the relation of dietary cholesterol and atheroma in chickens. Circulation 3, 116 to 119.
Poole, J. F. C., and Florey, H. W. (1958): Changes in the endothelium of the aorta and the behaviour of macrophages in experimental atheroma in rabbits. J. Pathol. Bacteriol. 75, 245 to 251.
Radhakrishnamurthy, B., Eggen, D. A., Kokatnur, Y., Jirge, S., Strong, J. P., and Berenfoot, G. S. (1975): Composition of connective tissue in aortas from rhesus monkeys during regression if dietinduced fatty streaks. Lab. Invest. 33, 136 to 146.
St. Clair, R. W., Clarkson, T. B., and Lofland, H. B. (1972): Effects of regression of atherosclerotic lesions on the content and esterification of cholesterol by cell-free preparations of pigeon aorta. Circulat. Res. 31, 664 to 671.
Still, W. J. S. (1974): The topography of cholesterol-induced fatty streaks. Exp. molec. Pathol. 20, 374 to 386.
Still, W. J. S., and Marriot, P. R. (1964): Comparative morphology of the early atherosclerotic lesion in man and cholesterol atherosclerosis in the rabbit. An electron microscopic study. J. Atheroscler. Res. 4, 373 to 386.
Stein, Y., Stein, O., and Shapiro, B. (1963): Enzymic pathways of glyceride and phospholipid synthesis in aortic homogenates. Biochim. Biophys. Acta 70, 33 to 42.
Vesselinovitch, D., Wissler, R. W., Hughes, R., and Borensztajn, J. (1976): Reversal of advanced atherosclerosis in rhesus monkeys. Part 1. Light microscopic studies. Atherosclerosis 23, 155 to 176.
Wilens, S. L. (1959): Regression of traumatic eutoneous and aural lipid deposits in cholesterol fed rabbits. Amer. J. Pathol. 35, 633 to 647.
Wolman, M., and Gaton, E. (1976): Macrophages and smooth muscle cells in the pathogenesis of atherosclerosis. J. Israel Med. Ass. 99, 450.
Zemplenyi, T. (1962): Enzymes of the arterial wall. J. Atheroscler. Res. 2, 2 to 24.
Zemplenyi, T. (1968): Enzyme Biochemistry of the Arterial Wall. pp. 164 to 167, 214 to 225, Lloyd-Luke, London.
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Adams, C.W.M. (1978). Some Cellular and Metabolic Aspects of Regression of Atherosclerosis, and the Compensatory Effect of Senile Arterial Dilatation. In: International Symposium State of Prevention and Therapy in Human Arteriosclerosis and in Animal Models. Abhandlungen der Rheinisch-Westfälischen Akademie der Wissenschaften. VS Verlag für Sozialwissenschaften, Wiesbaden. https://doi.org/10.1007/978-3-663-06754-2_18
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DOI: https://doi.org/10.1007/978-3-663-06754-2_18
Publisher Name: VS Verlag für Sozialwissenschaften, Wiesbaden
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