Zusammenfassung
In diesem, dem traumatisierten Knochen gewidmeten Kapitel, wird zunächst auf die normale und gestörte Knochenbruchheilung eingegangen, die einen sehr komplexen pathoanatomischen und pathophysiologischen Hintergrund hat. Es wird dabei auf die einen Bruchspalt umgebende Demineralisation als Ausdruck eines regionalen Akzellerationsphänomens hingewiesen, die manchmal schwer von einem Infekt abzugrenzen ist. Einen großen Teil dieses Kapitels nehmen die vielfältigen stressinduzierten Knochenveränderungen ein, angefangen von der Stressfraktur über chronische Abrissfrakturen bis hin zu tumorsimulierenden Stressphänomenen an den Enthesen. Mit stressinduzierten Skelettveränderungen sollte sich der Radiologe gut auskennen, denn der Stress auf den muskuloskelettalen Apparat hat in den letzten drei Jahrzehnten durch Sport, vor allem mangelhaft und falsch trainierter Amateure, und ungewohnte berufliche Aktivitäten in einem erschreckenden Maße zugenommen.
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Freyschmidt, J. (2016). Der traumatisierte Knochen. In: Skeletterkrankungen. Springer, Berlin, Heidelberg. https://doi.org/10.1007/978-3-662-48137-0_2
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