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Male Sexual Desire Disorder

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Clinical Uro-Andrology

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Abstract

In the literature hypoactive sexual desire is the most frequently studied entity in the male sexual desire domain. In this chapter it is extensively discussed. Moreover, some facets of male hyperactive sexual desire are discussed.

Hypoactive sexual desire is associated with multiple biological and psychological causes

Sexual desire is easily disturbed. Uncomplicated experience of sexual desire appears to demand that all physical and psychological systems involved function optimally. The fact that decreased sexual desire is a ‘final common pathway’ of a diverse range of causes has not contributed to stimulation of scientific research into these factors.

The number of properly controlled studies into pharmacological treatment of men with decreased sexual desire is very limited. There are no studies with control groups in the current literature on the psychological treatment of the problems men with hypoactive sexual desire suffer from. Cognitive behavioural therapy is possibly an effective approach; however, this observation has only been done in one single study in women.

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Notes

  1. 1.

    The term hypogonadism is used when the measured T levels are below 8 nmol/l.

  2. 2.

    MetS comprises a cluster of cardiovascular risk factors, such as type II diabetes mellitus, abdominal obesity, dyslipidemia and high blood pressure (AHA/NHLBI/ADA Conference Proceedings 2004). The mass of abdominal fat is the basis for the MetS. Abdominal fat performs endocrine activity: it converts the circulating T into the sex hormone estradiol (Diaz-Arjonilla et al. 2009). Furthermore, it excretes substances that cause a chronic inflammatory reaction in the vessel wall (atherosclerosis) (Matarese et al. 2007).

  3. 3.

    Klinefelter’s syndrome is a genetic condition in males in which the cells contain at least one extra X chromosome. The syndrome has multiple variations, of which the least complex is the 47, XXY karyotype, in which there are 47 chromosomes per cell of which XXY are the sex chromosomes. Therefore, this variation is also known as the XXY syndrome. The Klinefelter variations 48, XXXY and 49, XXXXY with even more extra X chromosomes have similar clinical presentations. The syndrome was first described by Harry Klinefelter in 1942. It occurs in 1 in 500 to 1,000 male births.

  4. 4.

    Kallmann syndrome starts before birth when a part of the brain that is used for olfaction is not formed. Because of this there also is no connection in the part of the brain between hypothalamus and pituitary. Consequentially the pituitary cannot receive any signals from the hypothalamus and therefore no LH and FSH are secreted. The consequence of the lack of LH secretion is that there is no production of sex hormones so that puberty does not commence. In addition, it causes infertility. The disorder occurs in 1 in 10,000 males and in approximately 1 in 70,000 females.

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Authors and Affiliations

  1. Department of Urology, VU University Medical Center, De Boelelaan 1117, Amsterdam, 1081 HV, The Netherlands

    Eric J. H. Meuleman

  2. Faculty of Psychology and Educational Sciences, Open University, 2960, Heerlen, 6401 DL, The Netherlands

    Jacques J. D. M. van Lankveld

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Correspondence to Eric J. H. Meuleman .

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  1. Urology, University of Naples AOU Federico II, Naples, Italy

    Vincenzo Mirone

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Meuleman, E.J.H., van Lankveld, J.J.D.M. (2015). Male Sexual Desire Disorder. In: Mirone, V. (eds) Clinical Uro-Andrology. Springer, Berlin, Heidelberg. https://doi.org/10.1007/978-3-662-45018-5_8

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