Abstract
In recent studies, anti-glomerular basement membrane antibodies (anti-GBM) have been implicated as causative agent in the pathogenesis of certain human glomerulonephritides [1]. This conclusion was based on the observation that anti-GBM antibodies are present in the kidneys of all cases of Goodpasture’s syndrome and one-half of the cases of human subacute glomerulonephritis so far examined. Glomerulonephritis can be passively transferred with anti-GBM antibodies, eluted from nephritic human kidneys.
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Hammer, D.K. (1969). Experimental Glomerulonephritis in Unresponsive Rabbits after Termination of Immunologic Tolerance. In: Westphal, O., Bock, HE., Grundmann, E. (eds) Current Problems in Immunology. Bayer-Symposium, vol 1. Springer, Berlin, Heidelberg. https://doi.org/10.1007/978-3-662-29237-2_27
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DOI: https://doi.org/10.1007/978-3-662-29237-2_27
Publisher Name: Springer, Berlin, Heidelberg
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