Abstract
Recent experimental studies show that the smooth muscle of the cavernous tissue plays a key role in the hemodynamics of penile erection [5, 8]. A fully rigid erection requires, in addition to cavernous relaxation, a large reduction in the cavernous outflow [2], which is brought about both by the passive compression of venous cushions between the cavernous sinusoids and the tunica albuginea [2], and the squeezing of the obliquely running venae perforantes within the tunica albuginea [7]. Therefore, excessive cavernous outflow can be caused by incomplete cavernosal relaxation, e.g., by cavernous fibrosis due to diabetes, or by disturbances of the squeezing mechanism within the tunica albuginea, or by both.
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© 1991 Springer-Verlag Berlin Heidelberg
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Stief, C.G. (1991). Evaluation of the Cavernous Occlusive System. In: Jonas, U., Thon, W.F., Stief, C.G. (eds) Erectile Dysfunction. Springer, Berlin, Heidelberg. https://doi.org/10.1007/978-3-662-00986-4_13
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DOI: https://doi.org/10.1007/978-3-662-00986-4_13
Publisher Name: Springer, Berlin, Heidelberg
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