Correlation Between Gadolinium-Enhanced MRI and Neurophysiology in Bell’s Palsy: A Preliminary Study
Fifty percent of patients with clinically complete Bell’s palsy run the risk of suffering from sequelae, including persisting muscle weakness and dyskinesia (abnormal facial muscle movements). The pathophysiological event that causes facial dyskinesia and the location of the primary nerve injury in Bell’s palsy is unknown. It has been speculated that central (facial nucleus) hyperexcitability, ephaptic transmission along the facial nerve, or defect reinnervation (to false muscle) of the nerve fibers may be responsible for facial dyskinesias . In the present study, gadolinium-enhanced MRI (GadMRI) and refined neurophysiological methods were used in an attempt to locate the primary nerve injury and get more information on the pathogenesis of facial dyskinesias in Bell’s palsy.
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