Hemodynamic Monitoring in Trauma

  • J. D. Edwards
Conference paper
Part of the Update in Intensive Care and Emergency Medicine book series (UICM, volume 11)

Abstract

Trauma remains the leading cause of preventable death in young adults [1]. Several recent reports in the UK have highlighted possible inadequacies in the provision of all aspects of traumacare-initial clinical assessment and resuscitation, definitive radiological investigation and surgery and provision of intensive care [2, 3]. Management of trauma victims from the cardiorespiratory point of view has until recently been dominated by the concept of an “ebb phase” of metabolic rate and oxygen consumption (VO2) which was said to last for the first 48 hrs after injury [4]. The therapeutic implication of this concept was never directly stated but was tacitly accepted by many clinicians to mean that in the early post-traumatic period normal values of cardiac output (CO) would suffice. This lead to the restoration of blood pressure and urine output as the goals of resuscitation. In fact the importance of achieving an adequate urine output before surgery in wounded soldiers was demonstrated by Ladd in the Korean conflict (see below) [5]. Even this simple message was unheard of by the majority of physicians not practising in trauma centres. The “ebb phase” concept has recently been questioned [6]. The human data on which it was based is unconvincing. The classic, much quoted series, was of only 7 patients with trivial injuries. These were studied after elective orthopaedic procedures. There is strong circumstantial evidence in the literature that an “ebb phase” does not exist - possibly the reverse.

Keywords

Catheter Depression Urea Lactate Creatinine 

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Copyright information

© Springer-Verlag Berlin Heidelberg 1991

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  • J. D. Edwards

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