Advertisement

The Histology of Rare Human Tumors and their Relationships to General Tumor Pathology

  • R. Laumonier
  • G. Chomette
Part of the Recent Results in Cancer Research book series (RECENTCANCER, volume 44)

Abstract

Certain human tumors may be considered rare in the light of certain facts:
  • the low percentage rate of certain cancers leads to a rare statistical level amongst tumors in general. This rarity is absolute for certain very rare cancers in all countries, and for all workers (such as the esthésioneurocytoma). It is relative for certain geographical distributions (hepatomas) and for age (example: adult nephroblastoma).

  • the morphological rarity is represented by the fact that some relatively rare tumors sometimes show unusual histological aspects. These may be undifferentiated forms of usually differentiated cancers (anaplastic forms of osteogenic sarcomas), or unusual differentiations (rare epidermoid carcinomas of the stomach). This group is the main supply of Nadal Masson’s “ignotomas” which are the concern and sometimes the confusion of pathologists (Masson 1968).

  • the rarity of their evolution is shown by the disconcerting behaviour of certain tumors with regard to their natural history. Either cancers which do not generally give rise to secondaries, develop visceral metastasis (such as the basal cell carcinoma of the skin). Or, an abnormally long evolution is found in patients with a very malignant tumor (melanomas, generalized carcinomas). Or, even spontaneous recovery is found (choriocarcinoma of the placenta, rare observations of glandular carcinomas).

Keywords

Basal Cell Carcinoma Medullary Carcinoma Tumor Clone Alveolar Rhabdomyosarcoma lymphOid Stroma 
These keywords were added by machine and not by the authors. This process is experimental and the keywords may be updated as the learning algorithm improves.

Preview

Unable to display preview. Download preview PDF.

Unable to display preview. Download preview PDF.

References

  1. 1.
    Amiel, J. L.: In: Cell bound immunity, 143, p. 197, Liège 1967.Google Scholar
  2. 2.
    Anthony, P. P., Drury, R. A.: J. clin. Path. 23, 110 (1970).PubMedCrossRefGoogle Scholar
  3. 3.
    Ashworth, C. T.: Book reviews, Cancer Res. 27, 1007 (1967).Google Scholar
  4. 4.
    Boyd, W.: Springfield: CT. Thomas Edit. 1966.Google Scholar
  5. 5.
    Dailly, R., Forthomme, J.: Presse méd. 73, 2867 (1965).PubMedGoogle Scholar
  6. 6.
    Delarue, J., Abelanet, A., Chomette, G.: Presse méd. 73, 1517 (1965).PubMedGoogle Scholar
  7. 7.
    Elston, C. W.: J. Path. Bact. 97, 261 (1969).CrossRefGoogle Scholar
  8. 8.
    Laumonier, R., Laquerriere, R., Marche, C.: Rev. franç. Etud. clin. biol. 8, 884 (1963).PubMedGoogle Scholar
  9. 9.
    Masson, P.: Paris: Maloine Edit. 1968.Google Scholar
  10. 10.
    Mathe, G.: Bull. Cancer 58, 33 (1967).Google Scholar
  11. 11.
    Payan, A.: Arch. Anat. path. 19, 153 (1971).Google Scholar
  12. 12.
    Peyron, A.: C. R. Soc. Biol. (Paris) 135, 281 (1941).Google Scholar
  13. 13.
    Peyron, A.: C. R. Soc. Biol. (Paris) 135, 347 (1941).Google Scholar
  14. 14.
    Spärck, J. V.: Acta path, microbiol. scand. 77, 24 (1969)CrossRefGoogle Scholar

Copyright information

© Springer-Verlag Berlin · Heidelberg 1973

Authors and Affiliations

  • R. Laumonier
  • G. Chomette

There are no affiliations available

Personalised recommendations