Abstract
The acute respiratory distress syndrome (ARDS), a disorder with a mortality of approximately 50–70%, is characterized by a sudden, diffuse or localized inflammation of the lung, which-in the further course-induces
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1)
a non-cardiogenic pulmonary edema,
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a pulmonary arterial hypertension,
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a reduction of total compliance of the lung, and
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4)
a progressive systemic hypoxemia due to a pulmonary ventilation/perfusion mismatching leading to an increased portion of intrapulmonary right-to-left shunt areas [1].
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Gerlach, H. (1995). Inhaled Nitric Oxide in ARDS: Just a Replacement Therapy?. In: Vincent, JL. (eds) Yearbook of Intensive Care and Emergency Medicine. Yearbook of Intensive Care and Emergency Medicine, vol 1995. Springer, Berlin, Heidelberg. https://doi.org/10.1007/978-3-642-79154-3_28
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DOI: https://doi.org/10.1007/978-3-642-79154-3_28
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