Abstract
There are many reports of poor correlation between clinical findings and somatosensory evoked potentials (SEP). This holds especially for patients with demyelinating disease, where pathological latency delays and amplitude reductions are common even in patients without sensory deficits or specific complaints. Until now there has been no sufficiently accepted explanation for this phenomenon [2, 3, 4]. The notion of the high sensitivity and low specificity of the SEP triggered the present study; therefore, we attempted to correlate somatosensory and SEP abnormalities and to provide new data to localize brain-stem lesions by means of the SEP.
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References
Anziska B, Cracco RQ (1980) Short latency somatosensory evoked potentials: studies in patients with focal neurological disease. Electroencephalogr. Clin Neurophysiol 49:227–239
Buettner UW (1991) Critical evaluation of somatosensory evoked potentials in multiple sclerosis. In: Wiethölter H, Dichgans J, Mertin J (eds) Current concepts in multiple sclerosis. Excerpta Medica, Elsevier, Amsterdam, pp 61–66
Chiappa KH (1990) Evoked potentials in clinical medicine. Raven Press, New York
Chiappa KH, Choi S, Young RR (1980) Short latency somatosensory evoked potentials following median nerve stimulation in patients with neurological lesions. In: Desmedt JE (ed) Progress in clinical neurophysiology, vol 7, Karger, Basel, pp 264–281
Dillmann U, Besser R, Eghbal R, Koehler J, Ludwig B (1990) SEP and MRI findings in patients with localized brain-stem lesions. In: P.M. Rossini, F. Mauguiere (eds) New trends and advanced techniques in clinical neurophysiology, EEG Suppl. 41. Elsevier, Amsterdam, pp 314–319
Halliday AM, Wakefield GW (1963) Cerebral evoked potentials in patients with dissociated sensory loss. J Neurol Neurosurg Psychiatry 26:211–219
Holmgren H, Leijon G, Boivie J, Johansson I, Ilievska L (1990) Central post-stroke pain– somatosensory evoked potentials in relation to location of the lesion and sensory signs. Pain 40:43–52
Mauguière F, Brunon AM, Echallier JF, Courjon J (1982) Early somatosensory evoked potentials in lesions of the lemniscal pathway in humans. In: Courjon J, Mauguière F, Revol M (eds) Clinical applications of evoked potentials in neurology. Raven Press, New York, pp 321–338
Sawada H, Seriu N, Udaka F, Kameyama M (1990) Magnetic resonance imaging of medial medullary infarction. Stroke 21:963–966
Tomberg C, Desmedt JE, Ozaki I, Noël P (1991) Nasopharyngeal recordings of somatosensory evoked potentials document the medullary origin of the N18 far-field. Electroencephalogr. Clin Neurophysiol 80:496–503
Yamada T, Kayamori R, Kimura J, Beck DO (1984) Topopgraphy of somatosensory evoked potentials after stimulation of the median nerve. Electroencephalogr. Clin Neurophysiol 59:29–43
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© 1993 Springer-Verlag Berlin · Heidelberg
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Claß, R., Buettner, U.W. (1993). Correlation of Somatosensory Evoked Potentials and Somatosensory Findings in Patients with Brain-Stem Lesions. In: Caplan, L.R., Hopf, H.C. (eds) Brain-Stem Localization and Function. Springer, Berlin, Heidelberg. https://doi.org/10.1007/978-3-642-78172-8_18
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DOI: https://doi.org/10.1007/978-3-642-78172-8_18
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