Abstract
The origin of CPV, more than a decade after its sudden appearance, still remains a mystery. Many hypothesis have been offered (Siegl 1984). Most are based upon CPV arising as a variant of a pre-existing parvovirus that has adapted to canines, either under field conditions or as a result of the selective pressure of attenuation, such as in the production of live vaccines. These hypotheses include:
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1.
Recombination of canine adenovirus with Binn’s minute virus of canines
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2.
Variation of MEV or an MEV vaccine strain
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3.
Variation of a field, vaccine or laboratory strain FPV
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4.
Derivation from an unidentified sylvatic reservoir
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© 1991 Springer-Verlag Berlin Heidelberg
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Vella, C., Ketteridge, S.W. (1991). Origins of Canine Parvovirus. In: Vella, C., Ketteridge, S.W. (eds) Canine Parvovirus: A New Pathogen. Springer, Berlin, Heidelberg. https://doi.org/10.1007/978-3-642-76797-5_5
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DOI: https://doi.org/10.1007/978-3-642-76797-5_5
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