Abstract
Bacterial translocation secondary to gut damage caused by traumatic shock is a source of posttraumatic sepsis. In several series, only one third of multiorgan failure patients with clinical manifestations of sepsis had an identifiable focus of infection (Goris 1985). The majority of ICU patients demonstrate the picture of clinical sepsis without a focus. In these patients, the gut frequently plays an important part as a shock organ involved in the subsequent development of sepsis.
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© 1991 Springer-Verlag Berlin, Heidelberg
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Schlag, G., Redl, H., Dinges, H.P., Davies, J., Radmore, K. (1991). Bacterial Translocation in a Baboon Model of Hypovolemic-Traumatic Shock. In: Schlag, G., Redl, H., Siegel, J.H., Traber, D.L. (eds) Shock, Sepsis, and Organ Failure. Springer, Berlin, Heidelberg. https://doi.org/10.1007/978-3-642-76511-7_3
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DOI: https://doi.org/10.1007/978-3-642-76511-7_3
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