Abstract
Aphasia is a common and frequently disabling symptom of neurological disease [1], but a great proportion or even the majority of initially aphasie patients recover eventually. Obviously, there are a number of important differences between the underlying pathophysiology of aphasia resulting from stroke in the acute and the chronic phase. Edema, ischemic penumbra, and diaschisis may cause deterioration of performance in the acute stage over and above that due to the actual loss of nervous tissue, and compensation and collateral vascularization may result in various degrees of improvement in the chronic period.
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© 1991 Springer-Verlag Berlin Heidelberg
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Wallesch, CW., Bak, T. (1991). Acute Aphasia. In: Hartmann, A., Kuschinsky, W., Hoyer, S. (eds) Cerebral Ischemia and Dementia. Springer, Berlin, Heidelberg. https://doi.org/10.1007/978-3-642-76208-6_27
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DOI: https://doi.org/10.1007/978-3-642-76208-6_27
Publisher Name: Springer, Berlin, Heidelberg
Print ISBN: 978-3-642-76210-9
Online ISBN: 978-3-642-76208-6
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