Differences Between Helicobacter pylori Associated Gastritis in Patients with Duodenal Ulcer, Pyloric Ulcer, Other Gastric Ulcer, and Gastritis Without Ulcer
Helicobacter (Campylobacter) pylori (HP) is the main causal agent of type B gastritis [1–3]. The spectrum of HP-associated pathology extends from gastritis in the absence of lesions, duodenal ulcer, and pyloric ulcer, to gastric ulcer . We undertook a study to establish whether, among these various groups, differences in the density of colonization by HP or in the degree or the activity of gastritis can be found.
KeywordsPeptic Ulcer Duodenal Ulcer Gastric Ulcer Antral Mucosa Ulcer Group
These keywords were added by machine and not by the authors. This process is experimental and the keywords may be updated as the learning algorithm improves.
Morris A, Nicholson G (1987) Ingestion of Campylobacter pyloridis causes gastritis and raised fasting gastric pH. Am J Gastroenterol 82:192–199.PubMedGoogle Scholar
Mahoney MJ, Wyatt JI, Littlewood JM (1988) Campylobacter pylori gastritis. Arch Dis Child 63:654–655.CrossRefGoogle Scholar
Stolte MS, Eidt S, Ritter M, Bethke B (1988) Campylobacter pylori und Gastritis — Association oder Induktion? Pathologe 10:21–26.Google Scholar
Börsch G (1987) Campylobacter pylori: new and renewed insights into gastritis associated ulcer disease (GAUD). Hepatogastroenterology 34:191–193.PubMedGoogle Scholar
Oi M, Oshida K, Sugimura S (1959) The location of gastric ulcers. Gastroenterology 36:45–56.PubMedGoogle Scholar
Oi M, Ito Y, Kumagi F, Yoshida K, Tanaka Y, Yoshikawa K, Miho O, Kilima M (1969) A possible dual control mechanism in the origin of peptic ulcer. Gastroenterology 57:280–293.PubMedGoogle Scholar
© Springer-Verlag Berlin Heidelberg 1990