Abstract
The secretory role of the submaxillary gland (SBG) has been sufficiently proven, as seen by the literature [1, 3, 5]. The morphological and functional supression of the SBG, achieved by surgically removing it, produces a syndrome classified as hypersialadenism [4]. We also verified that ablation of the SBG brings about changes in the whole organism (acting per se or interacting with other incretory glands), with a main role in the microvascularization (MV) of the endocrine system and the bone [2]. In this paper we wish to report the results of experiments in which the MV and clinical evolution of fracture callus were studied.
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References
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© 1990 Springer-Verlag Berlin Heidelberg
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Martínez, M.P.A., Gámez, F.N., Melón, R.M. (1990). Effects of Submaxillary Gland Ablation on Experimental Fracture: Callus and Its Microvascular Modifications. In: Arlet, J., Mazières, B. (eds) Bone Circulation and Bone Necrosis. Springer, Berlin, Heidelberg. https://doi.org/10.1007/978-3-642-73644-5_13
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DOI: https://doi.org/10.1007/978-3-642-73644-5_13
Publisher Name: Springer, Berlin, Heidelberg
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