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Direct Antitumor Effects of an LH-RH Agonist

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Abstract

Chronic administration of potent long-acting luteinizing hormone-releasing hormone (LH-RH) agonists to patients in sufficiently high doses causes a chemical castration through suppression of the pituitary-gonadal axis. This therapy appears effective in the treatment of hormone-dependent tumors, such as breast and prostate cancer (for updated results, see the chapters in this volume).

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© 1988 Springer-Verlag Berlin Heidelberg

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Foekens, J.A., Klijn, J.G.M. (1988). Direct Antitumor Effects of an LH-RH Agonist. In: Höffken, K. (eds) LH-RH Agonists in Oncology. Springer, Berlin, Heidelberg. https://doi.org/10.1007/978-3-642-73530-1_3

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  • DOI: https://doi.org/10.1007/978-3-642-73530-1_3

  • Publisher Name: Springer, Berlin, Heidelberg

  • Print ISBN: 978-3-642-73532-5

  • Online ISBN: 978-3-642-73530-1

  • eBook Packages: Springer Book Archive

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