Abstract
The ability of the inflammatory or nonspecific immune system to respond to antigenic stimuli is extremely important. Neutrophils (PMNs) are the leukocytes most responsive and capable, in the normal host, of controlling bacterial invasion. However, neutrophil dysfunction occurs after injury, including defects in chemotaxis [1], phagocytosis, and microbial killing [2]. In addition, depressed serum opsonic capacity and the presence of inhibitory substances further compromise neutrophil function in the burn victim [3].
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© 1989 Springer-Verlag, Berlin Heidelberg
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Gadd, M.A., Hansbrough, J.F. (1989). The Effect of Injury on Murine Neutrophil Oxidative Metabolism. In: Faist, E., Ninnemann, J.L., Green, D.R. (eds) Immune Consequences of Trauma, Shock, and Sepsis. Springer, Berlin, Heidelberg. https://doi.org/10.1007/978-3-642-73468-7_22
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DOI: https://doi.org/10.1007/978-3-642-73468-7_22
Publisher Name: Springer, Berlin, Heidelberg
Print ISBN: 978-3-642-73470-0
Online ISBN: 978-3-642-73468-7
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