Abstract
Splenic abscess is an unusual entity with an incidence of only 0.14%–0.70% in large autopsy series. Altemeier et al. (1973), for example, failed to find any cases in their review of 540 intraabdominal abscesses. The rarity of splenic infections can be explained by this organ’s phagocytic activity and its role in the immune system, which give it a unique capacity to resist local infection (San and Zuidema 1982). As early as 1938, Caldarera suggested that both an infectious agent (reaching the spleen by hematogenous spread or direct contact with a local focus of infection) and a pathologic spleen (defective function or architecture) are required to initiate splenic abscess.
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Anagnostoupoulos, C., Jacquenod, P., Chagnon, S., Bléry, M. (1988). Splenic Abscess and Infarction. In: Ultrasonography of the Spleen. Springer, Berlin, Heidelberg. https://doi.org/10.1007/978-3-642-73199-0_5
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DOI: https://doi.org/10.1007/978-3-642-73199-0_5
Publisher Name: Springer, Berlin, Heidelberg
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