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Part of the book series: NATO ASI Series ((ASIH,volume 20))

Abstract

There are two distinct ways in which amino acid supply could influence behaviour. Amino acid supply might control eating behaviour specifically, or eating and other behaviour might be modulated by effects of amino acid supply on neurotransmitters in the pathways organising such behaviour. Given that behaviour is organised by multisynaptic networks, it is extremely unlikely that an effect of precursors on transmitters would have a specific consequence for eating behaviour. If we allow realistically for the nature of behaviour and for the way the brain works, we must expect any control of eating behaviour itself by amino acid supply to be via some specific receptor system. If monoamine precursor metabolism were critical to such a sensor, then these widespread metabolic processes would have to be set within a specialisation that makes the operation of the sensor specific to eating behaviour, preventing it from affecting all the other physiology and behaviour that would be modulated by those same neurotransmitter effects elswhere in the brain or indeed the rest of the body.

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© 1988 Springer-Verlag Berlin Heidelberg

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Booth, D.A., Gibson, E.L. (1988). Control of Eating Behaviour by Amino Acid Supply. In: Huether, G. (eds) Amino Acid Availability and Brain Function in Health and Disease. NATO ASI Series, vol 20. Springer, Berlin, Heidelberg. https://doi.org/10.1007/978-3-642-73175-4_23

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  • DOI: https://doi.org/10.1007/978-3-642-73175-4_23

  • Publisher Name: Springer, Berlin, Heidelberg

  • Print ISBN: 978-3-642-73177-8

  • Online ISBN: 978-3-642-73175-4

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