Abstract
The last 15 years have seen rapid expansion in scientific enquiry into the neurobiological basis of Alzheimer’s Disease (AD). The new findings exhibit a promising cohesion and convergence of the evidence that has emerged from studies of different aspects of the disease and from observations during life and post mortem. It is also significant that structural lesions and the neurochemical deficits which have come into view during the past decade have proved highly selective, consistently affecting certain systems while leaving others intact. This suggests that there may be a unitary aetiological basis for the disease, of which the diverse neurobiological lesions are different expressions.
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Roth, M. (1987). Growing Points in the Neurobiology of Alzheimer’s Disease. In: Govoni, S., Battaini, F. (eds) Modification of Cell to Cell Signals During Normal and Pathological Aging. NATO ASI Series, vol 9. Springer, Berlin, Heidelberg. https://doi.org/10.1007/978-3-642-72729-0_16
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DOI: https://doi.org/10.1007/978-3-642-72729-0_16
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