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Variability of Fetal Heart Rate Deceleration, Blood Pressure, and Acid Base Alterations During Defined Repetitive Hypoxic Stress

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Fetal Heart Rate Monitoring

Abstract

Complete reduction of uterine blood flow (UBF) results in a decline of oxygen saturation (SO2) in the fetal arterial blood. Fetal heart rate (FHR) starts to decelerate when oxygen saturation falls about 10%–15% (Künzel et al. 1983) independent of the status of fetal oxygenation prior to the reduction of uterine blood flow. There was no critical limit of oxygen saturation for the induction of deceleration. The decrease in FHR is accompanied by a rise in blood pressure and respiratory and metabolic acidosis with considerable variability in quantity and in time (Künzel et al. 1983).

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References

  • Itskovitz J, Goetzmann BW, Rudolph AM (1982) The mechanism of late deceleration of the heart rate and its relationship to oxygenation in normoxemic and chronically hypoxemic fetal lambs. Am J Obstet Gynecol 142:66

    PubMed  CAS  Google Scholar 

  • Jensen A, Künzel W, Kastendieck E (1982) Epinephrine and norepinephrine release in the fetus after repeated hypoxia. J Perinat Med [Suppl 2] 10:109

    Article  Google Scholar 

  • Kastendieck E, Künzel W, Kurz CS (1980) Utilization of lactic acid and cardiovascular response in the sheep fetus receiving an infusion of lactic acid. Arch Gynäkol 230:21

    CAS  Google Scholar 

  • Kastendieck E, Paulick R, Martius J, Jensen A (1982) Einfluß der metabolischen Azidose auf Herzfrequenz, Blutdruck, Oxygenation, Säure-Basen-Status, Glucose- und Elektrolytkonzentrationen des Feten und Geschwindigkeit der fetalen Milchsäureelemination im chronischen Schaftexperiment. In: Dudenhausen JW, Saling E (Hrsg) Perinatale Medizin, Vol 9. Thieme, Stuttgart

    Google Scholar 

  • Künzel W, Kurz CS, Kastendieck E (1981) Die Variabilität der fetalen Herzfrequenzreaktion auf die Reduktion der uterinen Durchblutung. Z Geburtshilfe Perinat 185:343

    Google Scholar 

  • Künzel W, Kastendieck E, Hohmann E (1983) Heart rate and blood pressure response and metabolic changes in the sheep fetus following reduction of uterine blood flow. Gynecol Obstet Invest 15:300

    Article  PubMed  Google Scholar 

  • Moll W, Kastendieck E (1978) Accumulation and disappearance of lactate in a fetus with a hemochorial placenta. The role of placental transfer and fetal metabolism. J Perinat Med 6:246

    Article  PubMed  CAS  Google Scholar 

  • Parer JT, Krueger TR, Harris JL (1980) Fetal oxygen consumption and mechanisms of heart rate response during artificially produced late decelerations of fetal heart rate in sheep. Am J Obstet Gynecol 136:478

    PubMed  CAS  Google Scholar 

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© 1985 Springer-Verlag Berlin Heidelberg

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Kastendieck, E., Künzel, W., Jensen, A. (1985). Variability of Fetal Heart Rate Deceleration, Blood Pressure, and Acid Base Alterations During Defined Repetitive Hypoxic Stress. In: Künzel, W. (eds) Fetal Heart Rate Monitoring. Springer, Berlin, Heidelberg. https://doi.org/10.1007/978-3-642-70358-4_11

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  • DOI: https://doi.org/10.1007/978-3-642-70358-4_11

  • Publisher Name: Springer, Berlin, Heidelberg

  • Print ISBN: 978-3-642-70360-7

  • Online ISBN: 978-3-642-70358-4

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