Is Idiopathic Hyperaldosteronism a Variant of Essential Hypertension or of Conn’s Syndrome?

  • A. Lasaridis
  • D. L. Davies
  • C. Beretta-Piccoli
  • J. J. Brown
  • R. Fraser
  • C. Holloway
  • M. Ingram
  • A. F. Lever
  • J. I. S. Robertson
  • P. F. Semple
Conference paper
Part of the International Boehringer Mannheim Symposia book series (BOEHRINGER)


Primary hyperaldosteronism, or Conn’s syndrome, results from an aldosterone-producing adenoma of the adrenal cortex. It presents with features of mineralocorticoid excess — increased secretion of aldosterone, hypertension, hypernatraemia, increased exchangeable sodium, hypokalemia, low exchangeable potassium, and reduced plasma concentrations of renin and angiotensin II [1–3]. Removal of the tumour corrects the aldosterone excess, reverses the electrolyte abnormality, and often restores normal blood pressure [2]. Clinicians and pathologists have little difficulty in accepting the disease as an entity.


Essential Hypertension Primary Hyperaldosteronism Exchangeable Sodium Plasma Aldosterone Concentration Exchangeable Potassium 
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Copyright information

© Springer-Verlag Berlin Heidelberg 1983

Authors and Affiliations

  • A. Lasaridis
  • D. L. Davies
  • C. Beretta-Piccoli
  • J. J. Brown
  • R. Fraser
  • C. Holloway
  • M. Ingram
  • A. F. Lever
  • J. I. S. Robertson
  • P. F. Semple

There are no affiliations available

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