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Is Idiopathic Hyperaldosteronism a Variant of Essential Hypertension or of Conn’s Syndrome?

  • A. Lasaridis
  • D. L. Davies
  • C. Beretta-Piccoli
  • J. J. Brown
  • R. Fraser
  • C. Holloway
  • M. Ingram
  • A. F. Lever
  • J. I. S. Robertson
  • P. F. Semple
Conference paper
Part of the International Boehringer Mannheim Symposia book series (BOEHRINGER)

Abstract

Primary hyperaldosteronism, or Conn’s syndrome, results from an aldosterone-producing adenoma of the adrenal cortex. It presents with features of mineralocorticoid excess — increased secretion of aldosterone, hypertension, hypernatraemia, increased exchangeable sodium, hypokalemia, low exchangeable potassium, and reduced plasma concentrations of renin and angiotensin II [1–3]. Removal of the tumour corrects the aldosterone excess, reverses the electrolyte abnormality, and often restores normal blood pressure [2]. Clinicians and pathologists have little difficulty in accepting the disease as an entity.

Keywords

Essential Hypertension Primary Hyperaldosteronism Exchangeable Sodium Plasma Aldosterone Concentration Exchangeable Potassium 
These keywords were added by machine and not by the authors. This process is experimental and the keywords may be updated as the learning algorithm improves.

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Copyright information

© Springer-Verlag Berlin Heidelberg 1983

Authors and Affiliations

  • A. Lasaridis
  • D. L. Davies
  • C. Beretta-Piccoli
  • J. J. Brown
  • R. Fraser
  • C. Holloway
  • M. Ingram
  • A. F. Lever
  • J. I. S. Robertson
  • P. F. Semple

There are no affiliations available

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