Abstract
The functional anatomy of the mitral valve apparatus has been well documented in recent Years (2, 3, 13). Normal function of the mitral valve requires delicately co-ordinated movement of its components - the leaflets, chordae, papillary muscles, and the supporting annulus and left ventricular myocardium. The chordae tendineae serve an essential role in the mechanics of mitral valve closure during ventricular systole. Rupture of one or more leads to mitral regurgitation. The pathological description of ruptured chordae probably begins with Corvisart in 1812. Older publications have stressed bacterial endocarditis (often with pre-existing rheumatic heart disease), rheumatic heart disaease alone, trauma, myocardial infarction, and connective tissue disorders as the common causes (16, 20, 21, 24). Recently, much attention has been directed towards the “floppy” mitral valve as the cause of innocent clicks or murmurs, and for its association with chest pain, atrial and ventricular arrhythmias and/or sudden death in elderly people (5, 6).
Supported by Smith & Nephew Fellowship Grant
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© 1981 Springer-Verlag Berlin Heidelberg
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Zacharopoulos, L., Ross, J.K., Monro, J.L., Gallagher, P.J. (1981). Ruptured Mitral Chordae Tendineae: Surgical Experience in 102 Cases and Observations on the Valve Ultrastructure in this condition. In: Bircks, W., Ostermeyer, J., Schulte, H.D. (eds) Cardiovascular Surgery 1980. Springer, Berlin, Heidelberg. https://doi.org/10.1007/978-3-642-68172-1_4
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DOI: https://doi.org/10.1007/978-3-642-68172-1_4
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