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Tierexperimentelle Untersuchungen zum Einfluß von Dopamin und Urbason auf die Hämodynamik und Nierenlunktion im experimentellen Tourniquet-Schock

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Kreislaufschock

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Zusammenfassung

Nach Wiederdurchblutung akut ischämisch geschädigter Extremitäten kann es durch Anflutung kataboler Stoffwechselprodukte und Elektrolytentgleisung zum Tourniquet-Syndrom kommen (14, 25, 27). Hypotonie, Tachykardie, Oligo-Anurie, Hyperkaliämie und metabolische Azidose kennzeichnen das Vollbild des akuten Tourniquet-Schocks (15, 28, 29).

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Authors
  1. F. Ghussen
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  2. V. Beyerling
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  3. B. Bongartz
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  4. B. Grosse-Thie
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  5. W. Stock
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Editor information

Editors and Affiliations

  1. Institut für Anaesthesiologie der Freien Universität Berlin Klinikum Charlottenburg, Spandauer Damm 130, 1000, Berlin 19, Germany

    Jürgen Bernhard Brückner

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© 1980 Springer-Verlag Berlin Heidelberg

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Ghussen, F., Beyerling, V., Bongartz, B., Grosse-Thie, B., Stock, W. (1980). Tierexperimentelle Untersuchungen zum Einfluß von Dopamin und Urbason auf die Hämodynamik und Nierenlunktion im experimentellen Tourniquet-Schock. In: Brückner, J.B. (eds) Kreislaufschock. Anaesthesiologie und Intensivmedizin / Anaesthesiology and Intensive Care Medicine, vol 125. Springer, Berlin, Heidelberg. https://doi.org/10.1007/978-3-642-67443-3_20

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  • DOI: https://doi.org/10.1007/978-3-642-67443-3_20

  • Publisher Name: Springer, Berlin, Heidelberg

  • Print ISBN: 978-3-540-09660-3

  • Online ISBN: 978-3-642-67443-3

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