Zusammenfassung
Der koronare Gefäßwiderstand wird durch ein internes Kontrollsystem gesteuert, das mit sogenannter metabolischer Vasodilatation antwortet [5]. Diese kann z.B. aus dem mit zunehmender Herzarbeit verbundenen, erhöhten Herzstoffwechsel resultieren oder von reduzierter Sauerstoffzufuhr zum Herzen, wie bei der Hypoxie, verursacht werden. Dieser Regulationsmechanismus bleibt nach chirurgischer Denervation des Herzens ungestört [10]. Als verantwortlich für diese koronare Vasodilatation scheinen Adenosin und Prostaglandine zu sein [6].
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Ottesen, S., Renck, H., Vik-Mo, H., Jynge, P. (1980). Koronare Durchblutung bei lumbaler und thorakaler Periduralanalgesie. In: Wüst, H.J., Zindler, M. (eds) Neue Aspekte in der Regionalanaesthesie 1. Anaesthesiologie und Intensivmedizin / Anaesthesiology and Intensive Care Medicine, vol 124. Springer, Berlin, Heidelberg. https://doi.org/10.1007/978-3-642-67384-9_3
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