Abstract
The clinical result of cerebral hypoxia is caused by a complex interaction of different lesions which we shall try to describe separately. The variety of effects cerebral hypoxia may produce is due to the fact that it provokes specific reactions in all different cell types of the brain (Table).
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References
Ames, A., Wright, R.L., Kowada, M., Thurston, J.M., Majno, G.: Cerebral ischemia II. The no-reflow phenomenon. Amer. J. Path. 52, 437–453 (1968).
Garcia, J.: Personal communication.
Heiss, W.D., Gerstenbrand, F., Prosenz, T., Krein, J.: The prognostic value of cerebral blood flow measurement in patients with the apallic syndrome. J. Neurol. Sci. 16, 279–382 (1972).
Hekmatpanah, J.: Cerebral blood flow dynamics in hypertension and cardiac arrest. Neurology 23, 174–180 (1973).
Hossmann, K.A. and Zimmermann, V.: Factors influencing the recovery of the monkey brain after prolonged cerebral ischemia. In: Pathology of cerebral microcirculation (ed. J. Cervòs-Navarro), p. 354. Berlin-New York: Walter de Gruyter 1974.
Ingvar, D.H., Brun, A.: Das komplette apallische Syndrom. Arch. Psychiat. Nervenkr. 215, 219–239 (1972).
Johnston, I.H., Rowan, I.O., Harper, A.M., Jennett, W.B.: Raised intracranial pressure and cerebral blood flow. I. Cisterna magna infusion in primates. J. Neurol. Neurosurg. Psychiat. 35, 258–296 (1972).
Langfitt, T.W., Weinstein, J.D., Kassell, N.F.: Cerebral vasomotor paralysis produced by intracranial hypertension. Neurology (Minneap.) 15, 622–641 (1965).
Matakas, F., CERVòS-Navarro, J., Schneider, H.: Experimental brain death, I, Morphology and fine structure of the brain. J. Neurol. Neurosurg. Psychiat. 36, 497–508 (1973).
Matakas, F., von Waechter, R., Knüpling, R., Potolicchio, J.S.: Increase in cerebral perfusion pressure by arterial hypertension in brain swelling. J. Neurosurg. 42, 282–289 (1975).
Nelson, E., Kawamura, J., Sunaga, T., Rennels, M.L., Gertz, S.D.: Scanning-and transmission electron microscopic study of endothelial lesion following ischemia with special attention to ischemic and “normal” branch points. In: Pathology of Cerebral Microcirculation (ed. J. Cervòs-Navarro), pp. 267–273. Berlin-New York: Walter de Gruyter 1974.
Waltz, H.G., Sundt, T.M.: The microvasculature and microcirculation of the cerebral cortex after arterial occlusion. Brain 90, 681–696 (1967).
Wolff, J.: Über die Möglichkeit der Kapillarverengung im zentralen Nervensystem, Eine elektronenmikroskopische Studie an der Großhirnrinde des Kaninchens. Z. Zellforsch. 63, 593–611 (1964).
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Cervós-Navarro, J., Matakas, F. (1975). Neuropathology of Cerebral Hypoxia. In: Penzholz, H., Brock, M., Hamer, J. (eds) Brain Hypoxia. Advances in Neurosurgery, vol 3. Springer, Berlin, Heidelberg. https://doi.org/10.1007/978-3-642-66239-3_1
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DOI: https://doi.org/10.1007/978-3-642-66239-3_1
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