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Intracranial Pressure after Prolonged Cerebral Ischemia

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Intracranial Pressure II

Abstract

Brain swelling accompanies ischemic brain lesions. It is induced by ischemic inhibition of cell metabolism which causes an increase in brain osmolarity and a shift of ions and water from the extracellular into the intracellular compartment (1). When ischemia is complete, brain swelling is limited by the available fluid in the blood and CSF compartment, but considerable water influx occurs as soon as blood recirculation is restored (2).

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References

  1. Van Harreveld, A.: Brain Tissue Electrolytes. London: Butterworths 1966.

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  2. Hossmann, K.-A., Zimmermann, V.: CBF, brain water and electrolytes during recovery from 1 hour’s cerebral ischemia. In: Proceedings International Symposium on Cerebral Circulation and Metabolism, Philadelphia, 1973. In press.

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  3. Häggendal, E., Löfgren, J., Nilsson, N.J., Zwetnow, N.N.: Prolonged cerebral hyperemia after periods of increased cerebrospinal fluid pressure in dogs. Acta physiol. scand. 79, 272–279 (1970).

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  4. Hossmann, K.-A., Lechtape-Grüter, H., Hossmann, V.: The role of cerebral blood flow for the recovery of the brain after prolonged ischemia. Z. Neurol. 204, 281–299 (1973).

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© 1975 Springer-Verlag Berlin Heidelberg

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Zimmermann, V., Hossmann, V., Hossmann, KA. (1975). Intracranial Pressure after Prolonged Cerebral Ischemia. In: Lundberg, N., Pontén, U., Brock, M. (eds) Intracranial Pressure II. Springer, Berlin, Heidelberg. https://doi.org/10.1007/978-3-642-66086-3_37

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  • DOI: https://doi.org/10.1007/978-3-642-66086-3_37

  • Publisher Name: Springer, Berlin, Heidelberg

  • Print ISBN: 978-3-642-66088-7

  • Online ISBN: 978-3-642-66086-3

  • eBook Packages: Springer Book Archive

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