Zusammenfassung
Bei zerebral geschädigten Patienten besteht ein erhöhtes Risiko, aufgrund einer zerebralen Hypoxie bzw. Ischämie durch arterielle Hypoxämie, arterielle Hypotonie bzw. intrakranielle Hypertension eine sekundäre Hirnschädigung zu erleiden. Mögliche Folge des sekundären Hirnschadens ist die Verschlechterung der klinischen Prognose des Patienten (Miller u. Becker 1982).
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Sarrafzadeh, A., Keller, E. (1999). Oxygenierung und Metabolismus. In: Schwab, S., Krieger, D., Müllges, W., Hamann, G., Hacke, W. (eds) Neurologische Intensivmedizin. Springer, Berlin, Heidelberg. https://doi.org/10.1007/978-3-642-58415-2_15
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DOI: https://doi.org/10.1007/978-3-642-58415-2_15
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