Zusammenfassung
Die Gallenwegsmotilität dient der zeitgerechten Bereitstellung der Gallenflüssigkeit bei den Verdauungsvorgängen, der Aufrechterhaltung des enterohepatischen Kreislaufs und der Verhinderung von Stase, die zu Entzündung und Steinbildung disponiert. Der Fluß wird bestimmt durch die Gallesekretion der Leber, durch den Kontraktionszustand der Gallenblase, durch die Motilität des Oddi-Sphinkter und durch die Bewegungsvorgänge im Duodenum. Gallenblasendysfunktion, Postcholezystektomiesyndrom und Funktionsstörungen des Oddi-Sphinkter werden mit Motilitätsstörungen der Gallenwege in Zusammenhang gebracht. Bei vielen der Patienten läßt sich allerdings kein organischer Befund an den Gallenwegen oder den benachbarten Organen erheben, so daß oft ein Beweis für die Zusammengehörigkeit von Mißempfindungen und funktionellen Störungen des Gallenwegsystems nicht erbracht werden kann. Aus diesem Grund sollte die Indikation für eine invasive Funktionsdiagnostik in jedem Fall sehr sorgfältig überprüft werden.
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Literatur
Allescher HD, Neuhaus H, Hagenmüller F et al. (1990) Effect of N-Butyl-scopolamine on sphincter of Oddi motility in patients during routine ERCP — a manometric study. Endoscopy 22: 160–163
Beglinger C, Hildebrand G, Adler G et al. (1992) Postprandial control of gallbladder contraction and exocrine pancreatic secretion in man. Eur J Clin Invest 22: 827–834
Botoman VA, Kozarek RA, Novell LA et al. (1994) Long-term outcome after endoscopic sphincterotomy in patients with biliary colic and suspected sphincter of Oddi dysfunction. Gastrointest Endosc 40: 165–170
Darweesh RMA, Dodds WJ, Hogan WJ et al. (1988) Efficacy of quantitative hepatobiliary scintigraphy and fatty-meal sonography for evaluating patients with suspected partial common bile duct obstruction. Gastroenterology 94: 779–786
Desautels SG, Slivka A, Hutson WR et al. (1999) Postcholecystectomy Pain Syndrome: pathophysiology of abdominal pain in sphincter of Oddi type III. Gastroenterology 116: 900–905
Dodds WJ (1990) Biliary tract motility and its relationship to clinical disorders. Am J Roentgenol 155: 247–258
Dodds WJ, Groh WJ, Darweesh RMA et al. (1985) Sonographic measurement of gallbladder volume. Am J Roentgenol 145: 1009–1011
Drossman DA, Li Z, Andruzzi E et al. (1993) U.S. householder survey of functional gastrointestinal disorders. Prevalence, sociodemography, and health impact. Dig Dis Sci 38: 1569–1580
Fenster LF, Lonborg R, Thirlby RC et al. (1995) What symptoms does cholecystectomy cure? Insights from an outcome measurement project and review of the literature. Am J Surg 169: 533–538
Fiorucci S, Bosso R, Morelli A (1992) Erythromycin stimulates gallbladder emptying and motilin release by atropine-sensitive pathways. Dig Dis Sci 37: 1678–1684
Geenen JE, Hogan WJ, Dodds WJ et al. (1989) The efficacy of endoscopic sphincterotomy after cholecystectomy in patients with sphincter of Oddi dysfunction. N Engl J Med 320: 82–87
Hogan WJ, Geenen JE (1988) Biliary dyskinesia. Endoscopy 20: 179–183
Jonson G, Nilsson DM, Nilsson T (1991) Cystic duct remnants and biliary symptoms after cholecystectomy: A randomised comparison of two operative techniques. Eur J Surg 157: 583
Khuroo MS, Zargar SA, Yattoo GN (1992) Efficacy of nifedipin therapy in patients with Sphincter of Oddi dysfunction: a prospective, double-blind randomized placebocontrolled, cross-over trial. Br J Clin Pharmacol 33: 477–485
Klinge U (1992) Grundlagen der Cholelithiasis. Epidemiologie und Pathogenese der Cholelithiasis. Chir Gastroenterol 8 (Suppl 2): 6–14
Kozarek RA (1988) Balloon dilatation of the sphincter of Oddi. Endoscopy 20: 207–210
Krishnamurthy GT, Krishnamurthy S (1988) Nuclear hepatology: Where is it heading now? J Nuc Med 29: 1144–1149
Kubota K, Itoh K, Shibayama K et al. (1989) Papillary function of patients with juxtapapillary duodenal diverticulum. Scand J Gastroenterol 24: 140–144
La Morte WW (1993) Biliary motility and abnormalities associated with cholesterol cholelithiasis. Curr Opin Gastroenterol 9: 810–816
Leivonen MK, Halttunen JAA, Kivilaakso EO (1996) Duodenal diverticulum at endoscopic retrograde cholangiopancreatography, analysis of 124 patients. Hepatogastroenterol 43: 961–966
O’Donnell LJD, Fairclough PD (1993) Gall stones and gall bladder motility. Gut 34: 440–443
Pasricha PJ, Miskovsky EP, Kalloo AN (1994) Intrasphincteric injection of botulinum toxin for suspected sphincter of Oddi dysfunction. Gut 35: 1319–1321
Rhodes M, Lennard TWJ, Farndon JR et al. (1988) Cholecystokinin (CCK) provocation test: long-term follow-up after cholecystectomy. Br J Surg 75: 951–953
Rolny P (1997) Endoscopic bile duct stent placement as a predictor of outcome following endoscopic sphincterotomy in patients with suspected sphincter of Oddi dysfunction. Eur J Gastroenterol Hepatol 9: 467–471
Sand J, Nordbach I, Koskinen M et al. (1993) Nifedipine for suspected Type II Sphincter of Oddi dyskinesia. Am J Gastroenterol 35: 1301–1305
Schmitt Th, Seifert H, Dietrich CF et al. (1999) Propofolsedierung bei endoskopischer Manometrie des Sphincter Oddi. Z Gastroenterol 37: 219–227
Stolk MFJ, van Erpecum KJ, Smout AJPM et al. (1993) Motor cycles with phase III antrum are associated with high motilin levels and prolonged gallbladder emptying. Am J Physiol 264: G596–G600
Tarnasky PR, Palesch YY, Cunningham JT et al. (1998) Pancreatic stenting prevents pancreatitis after biliary sphincterotomy in patients with sphincter of Oddi dysfunction. Gastroenterology 115: 1518–1524
Thune A, Baker AR, Saccone GTP et al. (1990) Differing effects of pethidine and morphine on human sphincter of Oddi motility. Br J Surg 77: 992–995
Wehrmann T, Wiemer K, Lembcke B et al. (1995) Effect of endoscopic sphincterotomy on sphincter of Oddi manometry results in patients with or without papillary stenosis. Z Gastroenterol 33: 662–668
Wehrmann T, Aharonoff H, Dietrich CF et al. (1997 a) Does ultrasonography allow prediction of biliary sphincter of Oddi dysfunction? Z Gastroenterol 35: 449–457
Wehrmann T, Marek S, Hanisch E et al. (1997 b) Causes and management of recurrent biliary pain after successful nonoperative gallstone treatment. Am J Gastroenterol 92: 132–138
Yap L, Wycherley AG, Morphett AD et al. (1991) Acalculous biliary pain: Cholecystectomy alleviates symptoms in patients with abnormal cholescintigraphy. Gastroenterology 101: 700–703
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Wienbeck, M., Eberl, T. (2000). Motilitätsstörungen der Gallenwege. In: Adler, G., Beglinger, C., Manns, M.P., Müller-Lissner, S., Schmiegel, W. (eds) Klinische Gastroenterologie und Stoffwechsel. Springer, Berlin, Heidelberg. https://doi.org/10.1007/978-3-642-57194-7_17
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