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The Stress Response after Traumatic Brain Injury: Metabolic and Hormonal Aspects

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The Stress Response of Critical Illness: Metabolic and Hormonal Aspects

Abstract

The pathophysiology of TBI can be considered as a dual insult composed of primary and secondary injuries. Growing experimental and clinical evidence suggests that disturbances of cerebral energy metabolism are a key factor in the pathogenesis of secondary cerebral damages. In addition, hormonal dysfunction after TBI, such as adrenal insufficiency, vasopressin, growth hormone, or thyrothropin deficiency, can be associated with poor prognosis. A better understanding of energy metabolism and hormonaldisturbances after TBIis necessary to improve the care management at the early phase of TBI.

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Author information

Authors and Affiliations

  1. Service de Réanimation, Hôpital Pasteur 2, 30 Voie Romaine, Nice, Cedex 1, 06001, France

    Hervé Quintard & Carole Ichai

  2. CNRS UMR6097- Université Nice Sophia Antipolis, Nice, F-06000, France

    Hervé Quintard

  3. IRCAN (INSERM U1081, CNRS UMR 7284), Université Nice Sophia Antipolis, Nice, F-06000, France

    Carole Ichai

  4. Pôle Anesthésie Réanimation, Hôpital Michallon, CHU de Grenoble, Grenoble, F-38043, France

    Jean-Francois Payen

  5. Université Grenoble Alpes, Grenoble Institut des Neurosciences, Grenoble, F-38042, France

    Jean-Francois Payen

Authors
  1. Hervé Quintard
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  2. Carole Ichai
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  3. Jean-Francois Payen
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Correspondence to Jean-Francois Payen .

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Editors and Affiliations

  1. Brussels, Belgium

    Jean-Charles Preiser

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Quintard, H., Ichai, C., Payen, JF. (2016). The Stress Response after Traumatic Brain Injury: Metabolic and Hormonal Aspects. In: Preiser, JC. (eds) The Stress Response of Critical Illness: Metabolic and Hormonal Aspects. Springer, Cham. https://doi.org/10.1007/978-3-319-27687-8_16

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  • DOI: https://doi.org/10.1007/978-3-319-27687-8_16

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  • Publisher Name: Springer, Cham

  • Print ISBN: 978-3-319-27685-4

  • Online ISBN: 978-3-319-27687-8

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