Abstract
Thinking about causation in illness has a long and complicated philosophical and psychological history. Descartes’ idea that the mind and body are separate is manifested in how we think about illness. Despite the idea many of us hold—that the psychological and physical are integrated—we often feel we have to know (or, for that matter, can know) whether it is the mind or body that is responsible for disease. Medical professionals offer a stellar example of dealing with medical patients in a dualistic way. Patients whom are deemed to have a “real” illness are treated for their physical ailments, with relatively little emphasis on the psychological factors that could contribute to or exacerbate illness. On the other hand, there are patients whom physicians simply give up on—they tell patients that their illness is solely “in their head”—thus relegating them to a world of limited medical treatments and being outsourced to psychology or psychiatry. However, the mental health fields have their own dualistic biases. Especially early on, mainstream psychoanalysis has tended to focus on unconscious psychological dynamics that create or contribute to illness. It’s reasonable to assume that in all matters involving physical complaints, there is a complex feedback loop in which the body and emotions activate each other. As we will see, it may be the case that some illnesses are particularly sensitive to stress and that some people are biologically vulnerable to become ill when faced with emotional challenges. However, no matter the disease, there is a preponderance of evidence that psychological intervention helps to reduce healthcare visits and procedures and that some people actually do feel better, with a reduction in physical symptoms, when involved in psychotherapy. It may be the case that psychodynamic approaches have an advantage because of our involvement in bringing unconscious dynamics to light, but much more research is needed to understand which approaches are helpful and why. In the meantime, as therapists, we are confronted with gray areas all of the time in our work. For myself at least, I simply don’t know when I meet someone in my practice that will see benefit in physical symptoms as a result of interventions I might provide or suggest. So “gray areas” mean not really knowing the extent to which someone’s illness is influenced by conscious or unconscious emotions, and my sense has been that I need to tolerate a great deal of ambiguity in not taking sides when listening to someone with physical illness. I will discuss the idea of somatopsychogenesis and those conditions many of us learned to label as psychosomatic in our general mental health training. Yet, we now know that a number of illnesses that had been previously referred to as being caused by one’s mind are actually highly influenced by physical susceptibilities. Several physical problems are understood to be a result of a complicated interplay of psychology, neurophysiology, and the havoc stress hormones bring to bear on the body in those that seem biologically vulnerable.
No one is free who is a slave to his body
Seneca The Younger
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Notes
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In the 1990s, there was much less sensitivity regarding the understanding of physical tolerance to pain medication. At the time, tolerance was considered synonymous with addiction. Yet, it does seem that some people use narcotic medication to manage emotional distress.
References
Äärelä, E. (2008). Bodily symptoms and a psychoanalytic model of affect. Scandinavian Psychoanalytic Review, 31, 29–37.
Abbey, S. E., & Garfinkel, P. E. (1991). Neurasthenia and chronic fatigue syndrome: The role of culture in the making of a diagnosis. American Journal of Psychiatry, 148, 1638–1646.
Aisenstein, M., & Rappoport de Aisemberg, E. (2010). Preface. Psychosomatics Today. London: Karnac.
Alexander, F. (1936). Addenda to “the medical value of psychoanalysis”. Psychoanalytic Quarterly, 5, 548–559.
Baumeister, D., Akhtar, R., Ciufolini, S., Pariante, C. M., & Mondelli, V. (2015). Childhood trauma and adulthood inflammation: A meta-analysis of peripheral C-reactive protein, interleukin-6 and tumour necrosis factor-α. Molecular Psychiatry. doi:10.1038/mp.2015.67.
Breuer, J., & Freud, S. (1957). Studies on hysteria. New York: Basic Books. (Original work published 1895)
Callahan, P., & Tsouderos, T. (2010, December 8). Chronic lyme disease: A dubious diagnosis. Chicago Tribune.
Cannon, W. B. (1915). Bodily changes in pain, hunger, fear and rage. New York: D. Appleton.
Chrousos, G. P., & Gold, P. W. (1992). The concepts of stress and stress system disorders: Overview of physical and behavioral homeostasis. JAMA, 267(9), 1244–1252.
Cohen, S., Janicki-Deverts, D., Doyle, W. J., Miller, G. E., Frank, E., Rabin, B. S., et al. (2012). Chronic stress, glucocorticoid receptor resistance, inflammation, and disease risk. Proceedings of the National Academy of Science, 109(16), 5995–5999.
Cornell, W. F. (2015). Somatic experience in psychoanalysis and psychotherapy: In the expressive language of the living. New York: Routledge.
Fenichel, O. (1945). Nature and classification of the so-called psychosomatic phenomena. Psychoanalytic Quarterly, 14, 287–312.
Fink, G. (2009). Stress: Definition and history. In L. R. Squire (Ed.), Encyclopedia of neurological sciences (pp. 549–555). Oxford, England: Academic.
Freud, S. (1894/1985). Draft E. How anxiety originates. In: Masson J. M. (Ed.), The complete letters of Sigmund Freud to Wilhelm Fliess 1887–1904 (pp. 78–83). Cambridge, MA: Belknap.
Freud, S. (1913/2010). The interpretation of dreams (A. A. Brill, Trans.) (3rd ed.). New York: Macmillan (1913). Bartleby.com (2010).
Geiss, A., Rohleder, N., & Anton, F. (2012). Evidence for an association between an enhanced reactivity of interleukin-6 levels and reduced glucocorticoid sensitivity in patients with fibromyalgia. Psychoneuroendocrinology, 37, 671–684.
Gerson, C. D., Gerson, M. J., Chang, L., Corazziari, E. S., Dumitrascu, D., Ghoshal, U. C., et al. (2015). A cross-cultural investigation of attachment style, catastrophizing, negative pain beliefs, and symptom severity in irritable bowel syndrome. Neurogastroenterology and Motility, 27, 490–500.
Greenberg, T. M. (2014). Encyclopedia of the neurological sciences. In M. Aminoff & R. Daroff (Eds.), Abnormal illness behaviors, Article 1072. London: Elsevier.
Hartocollis, P. (2002). ‘Actual Neurosis’ and Psychosomatic Medicine: The Vicissitudes of an Enigmatic Concept. International Journal of Psychoanalysis, 83, 1361–1373.
Jaèn, C., & Dalton, P. (2014). Asthma and odors: The role of risk perception in asthma exacerbation. Journal of Psychosomatic Research, 77(4), 302–308.
Jaremka, L. M., Glaser, R., Loving, T. J., Malarkey, W. B., Stowell, J. R., & Kiecolt-Glaser, J. K. (2013). Attachment anxiety is linked to alterations in cortisol production and cellular immunity. Psychological Science, 24, 272–279.
Kalmakis, K. A., Meyer, J. S., Chiodo, L., & Leung, K. (2015). Adverse childhood experiences and chronic hypothalamic-pituitary-adrenal activity. Stress, 1–5.
Kelley, J. M., Lembo, A. J., Ablon, J. S., Villanueva, J. J., Conboy, L. A., Levy, R., et al. (2009). Patient and practitioner influences on the placebo effect in irritable bowel syndrome. Psychosomatic Medicine, 71(7), 789–797.
Kohutis, E. A. (2010). Concreteness, metaphor, and psychosomatic disorders: Bridging the gap. Psychoanalytic Inquiry, 30, 416–429.
McDougall, J. (1974). The psychosoma and the psychoanalytic process. International Review of Psychoanalysis, 1, 437–459.
McDougall, J. (1989). Theaters of the body. A psychoanalytic approach to psychosomatic illness. New York: W.W. Norton.
McWilliams, N. (2011). Psychoanalytic diagnosis: Understanding personality structure in the clinical process (2nd ed.). New York: The Guilford Press.
Miller, G. E., Chen, E., & Zhou, E. S. (2007). If it goes up, must it come down? Chronic stress and the hypothalamic-pituitary-adrenocortical axis in humans. Psychological Bulletin, 133(1), 25–45.
Muller, J. P. (1992). A re-reading of studies on hysteria: The Freud-Breuer break revisited. Psychoanalytic Psychology, 9, 129–156.
National Institutes of Health. (2002). Stress system malfunction could lead to serious, life-threatening disease. NIH Backgrounder. Retrieved April 25, 2015, from http://www.nih.gov/news/pr/sep2002/nichd-09.htm.
Nicolaides, N. C., Kyratzi, E., Lamprokostopoulou, A., Chouros, G. P., & Charmandari, E. (2015). Stress, the stress system and the role of glucocorticoids. Neuroimmunomodulation, 22, 6–19.
Papadopoulos, A. S., & Cleare, A. J. (2012). Hypothalamic–pituitary–adrenal axis dysfunction in chronic fatigue syndrome. Nature Reviews Endocrinology, 8, 22–32.
Petrelluzzi, K. F. S., Garcia, M. C., Petta, C. A., Grassi-Kassisse, D. M., & Spadari-Bratfisch, R. C. (2008). Salivary cortisol concentrations, stress and quality of life in women with endometriosis and chronic pelvic pain. Stress, 11(5), 390–397.
Schafer, S. M., Colloca, L., & Wager, T. D. (2015). Conditioned placebo analgesia persists when subjects know they are receiving a placebo. The Journal of Pain, 16(5):412–420. Retrieved April 26, 2015, from http://www.jpain.org/article/S1526-5900(15)00033-4/pdf.
Selye, H. (1998). A syndrome produced by diverse nocuous agents. The Journal of Neuropsychiatry and Clinical Neurosciences, 10(2), 230–231. (Original work published 1936)
Sloate, P. (2010). Superego and sexuality: An analysis of a psychosomatic solution. Psychoanalytic Inquiry, 30, 457–473.
Smith, S. M., & Vale, W. W. (2006). The role of the hypothalamic-pituitary-adrenal axis in neuroendocrine responses to stress. Dialogues in Clinical Neurosciences, 8, 383–395.
Ulman, R. E., & Brothers, D. (1988). The shattered self: A psychoanalytic study of trauma. New York: The Analytic Press.
Wang, H., Weber, A., Schiltenwolf, M., & Amelung, D. (2014). Attachment style and cytokine levels in patients with fibromyalgia. A prospective longitudinal study. Schmerz, 28(5), 504–512.
Winnicott, D. W. (1966). Psycho-somatic illness in its positive and negative aspects. International Journal of Psychoanalysis, 47, 510–516.
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Greenberg, T.M. (2016). Gray Areas: When Illness May Be Particularly Impacted by Psychological Variables. In: Psychodynamic Perspectives on Aging and Illness. Springer, Cham. https://doi.org/10.1007/978-3-319-24289-7_5
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