Abstract
We have on average one million nephrons and they’re fully formed before we’re born. Premature birth and low birth weight coincides with fewer nephrons. No new nephrons are formed after birth and we lose up to 6,000 of them each year, making old age and low birth weight a risk factor for kidney disease.
Access this chapter
Tax calculation will be finalised at checkout
Purchases are for personal use only
References
Queisser-Luft A, Stolz G, Wiesel A, et al. Malformations in newborn: results based on 30,940 infants and fetuses from the Mainz congenital birth defect monitoring system (1990–1998). Arch Gynecol Obstet. 2002;266:163.
Sanna-Cherchi S, Caridi G, Weng PL, Scolari F, Perfumo F, Gharavi AG, Ghiggeri GM. Genetic approaches to human renal agenesis/hypoplasia and dysplasia. Pediatr Nephrol. 2007;22(10):1675.
Acknowledgement
1. Gray’s Anatomy 35th edition
2. Human Embryology – William J. Larsen, 3rd Edition
3. Medical Embryology – T.W. Sadler, 11th Edition
Author information
Authors and Affiliations
Corresponding author
Editor information
Editors and Affiliations
Rights and permissions
Copyright information
© 2016 Springer International Publishing Switzerland
About this chapter
Cite this chapter
Pandey, R. (2016). Development of Renal Tissues in First Twenty Weeks. In: Bhattacharya, N., Stubblefield, P. (eds) Human Fetal Growth and Development. Springer, Cham. https://doi.org/10.1007/978-3-319-14874-8_31
Download citation
DOI: https://doi.org/10.1007/978-3-319-14874-8_31
Published:
Publisher Name: Springer, Cham
Print ISBN: 978-3-319-14873-1
Online ISBN: 978-3-319-14874-8
eBook Packages: MedicineMedicine (R0)