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Abstract

Spasmodic dysphonia (SD) is a focal laryngeal dystonia characterized by involuntary, action-induced spasms of the muscles controlling vocal fold motion. The laryngeal adductor muscles (lateral cricoarytenoid [LCA], interarytenoid, and possibly the cricothyroid and thyroarytenoid [TA]), abductor muscle (posterior cricoarytenoid [PCA]), or rarely both groups of muscles may be affected. Adductor SD is characterized by a harsh, strangled, or effortful voice (glottal fry) with irregular phonatory breaks secondary to vocal fold hyperadduction or spasm. The supraglottic structures may be hyperfunctional as well. Abductor SD presents as a breathy, effortful, hypophonic voice with irregular breaks following consonant voicing secondary to vocal fold hyperabduction. Although the exact etiology of SD is unclear, SD is now recognized as a neurological disorder of central processing.

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Schwartz, J.S., Song, P., Blitzer, A. (2007). Spasmodic Dysphonia. In: Cooper, G. (eds) Therapeutic Uses of Botulinum Toxin. Humana Press. https://doi.org/10.1007/978-1-59745-247-2_8

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  • DOI: https://doi.org/10.1007/978-1-59745-247-2_8

  • Publisher Name: Humana Press

  • Print ISBN: 978-1-58829-914-7

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