Urodynamics (UDS) is the dynamic study of the storage and evacuation of urine. Its role is to provide the clinician with information about a patient’s lower urinary tract function. Ideally, the patient’s symptoms are reproduced during the study to provide objective measurements which correlate with the patient’s symptoms. UDS should be considered as an adjunct to a detailed history and physical exam to better characterize the clinical picture and facilitate optimal treatment planning. An important portion of the UDS is the bladder filling and storage phase. Involuntary detrusor contractions (IDCs) may occur during this portion of the study and may provide insight into the pathophysiology of the patient’s complaints. This chapter will focus on the different UDS manifestations of IDCs by investigating the UDS tracings of neurogenic and idiopathic detrusor overactivity, stress-induced overactivity, phasic overactivity, detrusor overactivity incontinence, detrusor after-contractions, and terminal detrusor overactivity.
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Haylen BT, et al. An International Urogynecological Association (IUGA)/International Continence Society (ICS) joint report on the terminology for female pelvic floor dysfunction. Neurourol Urodyn. 2010;29(1):4–20.PubMedGoogle Scholar
McGuire EJ, et al. Prognostic value of urodynamic testing in myelodysplastic patients. J Urol. 1981;126(2):205–9.PubMedGoogle Scholar
Buckner SA, et al. Spontaneous phasic activity of the pig urinary bladder smooth muscle: characteristics and sensitivity to potassium channel modulators. Br J Pharmacol. 2002;135(3):639–48.PubMedCentralPubMedCrossRefGoogle Scholar
Abrams P, et al. The standardisation of terminology of lower urinary tract function: report from the Standardisation Sub-committee of the International Continence Society. Neurourol Urodyn. 2002;21(2):167–78.PubMedCrossRefGoogle Scholar
Hindmarsh JR, Gosling PT, Deane AM. Bladder instability. Is the primary defect in the urethra? Br J Urol. 1983;55(6):648–51.PubMedCrossRefGoogle Scholar
Ostergard DR. The neurological control of micturition and integral voiding reflexes. Obstet Gynecol Surv. 1979;34(6):417–23.PubMedCrossRefGoogle Scholar
Jung SY, et al. Urethral afferent nerve activity affects the micturition reflex; implication for the relationship between stress incontinence and detrusor instability. J Urol. 1999;162(1):204–12.PubMedCrossRefGoogle Scholar
Bump RC, et al. Mixed urinary incontinence symptoms: urodynamic findings, incontinence severity, and treatment response. Obstet Gynecol. 2003;102(1):76–83.PubMedCrossRefGoogle Scholar
Webster GD, Kreder KJ. Voiding dysfunction following cystourethropexy: its evaluation and management. J Urol. 1990;144(3):670–3.PubMedGoogle Scholar
Smith AL, et al. Detrusor overactivity leak point pressure in women with urgency incontinence. Int Urogynecol J. 2012;23(4):443–6.PubMedCrossRefGoogle Scholar
Valentini FA, et al. Phasic or terminal detrusor overactivity in women: age, urodynamic findings and sphincter behavior relationships. Int Braz J Urol. 2011;37(6):773–80.PubMedCrossRefGoogle Scholar
Guralnick ML, et al. Objective differences between overactive bladder patients with and without urodynamically proven detrusor overactivity. Int Urogynecol J. 2010;21(3):325–9.PubMedCrossRefGoogle Scholar